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Postoperative emesis after laparoscopic pyloromyotomy in infantile hypertrophic pyloric stenosis
C Castellani1, T Peschaut, M Schippinger
1Department of Paediatric- and Adolescent Surgery, Medical University Graz, Graz, Austria.
Insights
Postoperative emesis in neonates after pyloric stenosis surgery can stem from infections, rapid feeding, or reflux. Reoperation for incomplete surgery is often difficult to confirm.
Area of Science:
- Pediatric Surgery
- Gastroenterology
- Neonatal Care
Background:
- Infantile hypertrophic pyloric stenosis (IHPS) is a common neonatal surgical condition.
- Laparoscopic pyloromyotomy (LP) is the standard surgical treatment for IHPS.
- Postoperative emesis (PE) is a common complication following LP.
Purpose of the Study:
- To investigate the causes of postoperative emesis (PE) in neonates who underwent laparoscopic pyloromyotomy (LP) for infantile hypertrophic pyloric stenosis (IHPS).
Main Methods:
- Retrospective review of a hospital database from 2000-2010.
- Inclusion of infants diagnosed with IHPS who underwent LP.
- Statistical analysis of clinical data including feeding protocols and diagnostic tests.
Main Results:
- Out of 43 infants meeting criteria, 21 (48.8%) experienced PE.
- PE was significantly more frequent in the fast-track feeding protocol (FTFP) group (12/15) compared to the conventional slow feeding protocol (CSFP) group (8/21).
- Enteric infections and gastro-oesophageal reflux (GER) were identified causes; reoperations for suspected incomplete myotomy were challenging to confirm.
Conclusions:
- Postoperative emesis in IHPS after LP necessitates thorough evaluation for diverse causes.
- Enteric viral infections, aggressive feeding strategies (FTFP), and GER are significant contributors to PE.
- Challenging clinical decisions arise when considering reoperation for incomplete pyloromyotomy due to PE.
Aim:
This study aimed to determine the causes of postoperative emesis (PE) in neonates with infantile hypertrophic pyloric stenosis (IHPS) after laparoscopic pyloromyotomy (LP).
Methods:
Retrospective review of the hospital database for infants with IHPS managed between 2000 and 2010 was performed. Relevant data were collected from the clinical records in the Medocs(®) system and used for statistical analysis.
Results:
During the 10-year period, 95 patients with IHPS were identified and 43 (36 boys and seven girls) fulfilled the inclusion criteria. PE occurred in 21 infants (48.8%), of which nine presented with manifestations of enteric infections, with confirmed diagnosis in four. PE was significantly higher in the fast track feeding protocol (FTFP) group 12/15 compared with the conventional slow feeding protocol (CSFP) group 8/21 (p = 0.019). Gastro-oesophageal reflux (GER) confirmed by 24-h impedance monitoring was responsible for PE in three. Operative revision for suspected incomplete pyloromyotomy was performed in five infants. However, incomplete myotomy could only be confirmed in two infants during surgery.
Conclusion:
Postoperative emesis in IHPS after LP requires careful evaluation as it can be a result of enteric viral infections, aggressive feeding protocols or GER. Decisions to perform reoperations for incomplete myotomy after LP due to PE are challenging.
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