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Related Concept Videos

Techniques of therapeutic communication I: Active Listening, Sharing Observations, Validation, and Using Touch01:15

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Tactile senses encompass touch, temperature, and pain, each mediated by specific receptors. Touch receptors detect mechanical energy or pressure against the skin. Sensory fibers from these receptors enter the spinal cord and relay information to the brain stem. Here, most fibers cross over to the opposite side of the brain. The touch information then moves to the thalamus, which projects a map of the body's surface onto the somatosensory areas of the parietal lobes in the cerebral cortex.
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Updated: May 2, 2026

A Labor-saving and Repeatable Touch-force Signaling Mutant Screen Protocol for the Study of Thigmomorphogenesis of a Model Plant Arabidopsis thaliana
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Touch me not.

Paras Karmacharya1, Kalpana Shah2, Ranjan Pathak1

  • 1Internal Medicine Department, Reading Health System, West Reading, PA, USA.

Journal of Community Hospital Internal Medicine Perspectives
|March 6, 2014
PubMed
Summary
This summary is machine-generated.

Central Poststroke Pain syndrome (CPSP) is a challenging condition resulting from brain pathway damage. Treatment focuses on reducing neuronal hyperexcitability, as traditional painkillers are often ineffective.

Keywords:
Central Poststroke PainDéjerine Roussy syndromegabapentinneuropathic painthalamic pain syndrome

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Area of Science:

  • Neurology
  • Neuroscience
  • Pain Medicine

Background:

  • Central Poststroke Pain (CPSP) arises from damage to the brain's somatosensory pathways.
  • Lesions can occur at various levels, including the thalamus, medulla, and cerebral cortex.
  • CPSP presents with sensory abnormalities and hyperesthesia linked to the lesion site.

Observation:

  • CPSP management is frequently difficult.
  • Standard analgesics typically show limited efficacy for CPSP.
  • Neuronal hyperexcitability is a key feature of this condition.

Findings:

  • First-line treatments target neuronal hyperexcitability.
  • Amitriptyline and lamotrigine are examples of first-line drugs.
  • Gabapentin is considered a second-line treatment option.

Implications:

  • Effective CPSP treatment requires addressing underlying neuronal mechanisms.
  • Pharmacological strategies focus on modulating hyperexcitable neurons.
  • Understanding lesion-pathway correlation aids in targeted therapeutic approaches.