The return of a disappearing entity: Dressler's syndrome after transvenous pacemaker implantation
António Tralhão1, Diogo Cavaco, Marisa Trabulo
1Department of Cardiology, Hospital de Santa Cruz, Centro Hospitalar de Lisboa Ocidental, Carnaxide, Portugal.
Insights
A rare Dressler-like syndrome developed in a patient after biventricular pacemaker implantation complicated by coronary sinus vein perforation. Steroid and colchicine therapy led to remission of pericardial and pleural effusions.
Area of Science:
- Cardiology
- Internal Medicine
- Medical Complications
Background:
- A 76-year-old woman with idiopathic dilated cardiomyopathy and left bundle branch block underwent biventricular pacemaker implantation.
- The procedure was complicated by coronary sinus tributary vein perforation, leading to pericardial effusion without tamponade.
Observation:
- Three months post-procedure, the patient presented with fever, pleuritic chest pain, and worsening functional class.
- Clinical examination revealed new pericardial and pleural effusions.
- Blood and pleural fluid cultures were sterile, and the patient did not respond to antibiotic therapy.
Findings:
- A Dressler-like syndrome was hypothesized as the cause of the effusions.
- The patient showed clinical improvement following a short course of steroid therapy.
- Discharge on colchicine resulted in complete remission of pleural effusion within two months, as confirmed by chest X-ray.
Implications:
- This case highlights a rare post-procedural complication mimicking Dressler's syndrome.
- It underscores the importance of considering inflammatory post-pericardiotomy syndromes in patients with unexplained effusions after cardiac procedures.
- Successful management with steroids and colchicine suggests these agents may be effective in similar cases.
Abstract:
A 76-year-old woman with idiopathic dilated cardiomyopathy and left bundle branch block was admitted for biventricular pacemaker implantation. The procedure was complicated by perforation of a coronary sinus tributary vein and ensuing pericardial effusion without tamponade. Three months later, she presented with fever, pleuritic chest pain and functional class worsening. Pericardial and pleural effusions were noted. Sterile blood and pleural fluid cultures and failure to respond to antibiotic therapy led to the hypothesis of a Dressler-like syndrome. The patient improved clinically with a short course of steroid therapy and was discharged on colchicine. A chest X-ray performed after 2 months showed a complete remission of pleural effusion.
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