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Updated: May 1, 2026

A Murine Model of Ischemic Retinal Injury Induced by Transient Bilateral Common Carotid Artery Occlusion
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Sequential bilateral retinal artery occlusion.

Noel Padrón-Pérez1, Janny Rosario Aronés2, Silvia Muñoz1

  • 1Department of Ophthalmology, Hospital Universitari de Bellvitge, Barcelona, Spain.

Clinical Ophthalmology (Auckland, N.Z.)
|April 22, 2014
PubMed
Summary
This summary is machine-generated.

A patient experienced sudden vision loss in both eyes due to atherogenic embolic events. Investigations revealed carotid artery stenosis and mitral valve calcification, highlighting the need for comprehensive vascular assessment.

Keywords:
Hollenhorst plaquebranch retinal artery occlusioncentral retinal artery occlusionloss of vision

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Area of Science:

  • Ophthalmology
  • Cardiology
  • Neurology

Background:

  • Sudden vision loss can indicate serious underlying vascular conditions.
  • Central retinal artery occlusion (CRAO) and branch retinal artery occlusion (BRAO) present as painless monocular vision loss.

Observation:

  • An 86-year-old woman presented with rapid, sequential bilateral vision loss.
  • Initial presentation included branch retinal artery occlusion (BRAO) in the right eye and central retinal artery occlusion (CRAO) with cilioretinal artery sparing in the left eye.

Findings:

  • Bilateral atherogenic embolic events were suspected.
  • Supra-aortic Doppler ultrasonography showed bilateral internal carotid artery stenosis.
  • Transthoracic echocardiography revealed severe mitral valve calcification with stenosis.

Implications:

  • This case underscores the importance of investigating embolic sources in patients with CRAO and BRAO.
  • Comprehensive vascular evaluation, including carotid and cardiac assessments, is crucial for identifying and preventing future embolic events.
  • Giant cell arteritis should be considered and excluded in patients over 50 presenting with CRAO.