Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Diphtheria01:28

Diphtheria

175
Diphtheria is an acute, toxin-mediated infectious disease that primarily affects the upper respiratory tract. It is caused by Corynebacterium diphtheriae, a Gram-positive, pleomorphic rod that lacks spore-forming capability and exhibits a characteristic club-shaped morphology under microscopic examination. While C. diphtheriae can asymptomatically colonize mucosal surfaces, clinical disease manifests only when the bacterial strain is lysogenized by a specific β-corynephage. This phage...
175
Development of the Oral Microbiota01:28

Development of the Oral Microbiota

65
The establishment of the oral microbiome begins before birth, challenging the long-held belief that the fetal oral cavity is sterile. The presence of oral microbes such as Streptococcus and Fusobacterium in amniotic fluid suggests that microbial exposure may occur in utero, potentially through translocation from the maternal oral or gastrointestinal tract. This early colonization primes the neonatal immune system and sets the stage for subsequent microbial succession. Maternal health,...
65
Pyloric Obstruction01:11

Pyloric Obstruction

40
Pyloric obstruction, also referred to as gastric outlet obstruction, is a condition characterized by narrowing or blockage at the pylorus—the muscular valve regulating the flow of stomach contents into the duodenum. When this passage becomes impaired, the stomach cannot effectively empty its contents into the small intestine. This disruption leads to a range of gastrointestinal symptoms, including early satiety, bloating, epigastric pain, postprandial nausea, persistent vomiting, and...
40
Prevention of Further Absorption of Poison01:14

Prevention of Further Absorption of Poison

1.4K
In cases of acute poisoning, the primary objective is to prevent further absorption of the toxic substance into the body. Immediate interventions using various decontamination techniques targeting the gastrointestinal (GI) tract can achieve this. Decontamination is crucial to prevent poison from entering the systemic circulation, which involves washing affected areas with water and mild soap and removing contaminated clothing. Once external decontamination is done, attention must be turned to...
1.4K
Development of Human Microbiota01:30

Development of Human Microbiota

61
The human microbiota begins developing at birth and undergoes continual change as we age. Infancy marks a critical period of microbial sensitivity, offering a “window of opportunity” during which beneficial microbes help mature the immune system. By age three, children typically develop a more stable and diverse microbial community. Newborns acquire microbes from their immediate environment; vaginal delivery favors maternal vaginal microbes, while cesarean births favor microbes from...
61
Bacterial Toxins01:12

Bacterial Toxins

148
Bacterial toxins are sophisticated virulence factors that enable pathogenic bacteria to interact with, invade, and damage host tissues. These toxins fall broadly into two types: protein exotoxins, which are secreted into the environment and target specific host receptors, and lipopolysaccharide endotoxins, which are structural components of the bacterial outer membrane released primarily during bacterial lysis or membrane shedding. Exotoxins generally act more selectively, binding to cell...
148

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

Nutritional status of neurologically impaired children: Impact on comorbidity.

Archives de pediatrie : organe officiel de la Societe francaise de pediatrie·2019
Same author

Baseline gut microbiota predicts clinical response and colitis in metastatic melanoma patients treated with ipilimumab.

Annals of oncology : official journal of the European Society for Medical Oncology·2019
Same author

Enteropathogens in paediatric gastroenteritis: comparison of routine diagnostic and molecular methods.

Clinical microbiology and infection : the official publication of the European Society of Clinical Microbiology and Infectious Diseases·2019
Same author

Rotavirus: the guard dies, but it does not surrender.

Infectious diseases (London, England)·2018
Same author

Relation between broiler and human Campylobacter jejuni strains isolated in Belgium from 2011 to 2013.

Journal of applied microbiology·2018
Same author

Immunome differences between porcine ileal and jejunal Peyer's patches revealed by global transcriptome sequencing of gut-associated lymphoid tissues.

Scientific reports·2018

Related Experiment Video

Updated: Apr 30, 2026

Isolation and Quantification of Botulinum Neurotoxin From Complex Matrices Using the BoTest Matrix Assays
12:25

Isolation and Quantification of Botulinum Neurotoxin From Complex Matrices Using the BoTest Matrix Assays

Published on: March 3, 2014

15.6K

[Infant botulism after honey exposure].

V Godart1, B Dan2, G Mascart3

  • 1Unité de pédiatrie générale, hôpital universitaire des enfants Reine-Fabiola, université Libre de Bruxelles, Bruxelles, Belgique.

Archives De Pediatrie : Organe Officiel De La Societe Francaise De Pediatrie
|April 29, 2014
PubMed
Summary

Infant botulism, a rare neuroparalytic disease, is caused by Clostridium botulinum neurotoxin. Honey exposure poses risks to infants under one year, necessitating avoidance to prevent this serious condition.

More Related Videos

Evaluating the Effect of Environmental Chemicals on Honey Bee Development from the Individual to Colony Level
07:39

Evaluating the Effect of Environmental Chemicals on Honey Bee Development from the Individual to Colony Level

Published on: April 1, 2017

8.6K
Effect of Hyaluronic Acid 35 kDa on an In Vitro Model of Preterm Small Intestinal Injury and Healing Using Enteroid-Derived Monolayers
09:36

Effect of Hyaluronic Acid 35 kDa on an In Vitro Model of Preterm Small Intestinal Injury and Healing Using Enteroid-Derived Monolayers

Published on: July 28, 2022

1.8K

Related Experiment Videos

Last Updated: Apr 30, 2026

Isolation and Quantification of Botulinum Neurotoxin From Complex Matrices Using the BoTest Matrix Assays
12:25

Isolation and Quantification of Botulinum Neurotoxin From Complex Matrices Using the BoTest Matrix Assays

Published on: March 3, 2014

15.6K
Evaluating the Effect of Environmental Chemicals on Honey Bee Development from the Individual to Colony Level
07:39

Evaluating the Effect of Environmental Chemicals on Honey Bee Development from the Individual to Colony Level

Published on: April 1, 2017

8.6K
Effect of Hyaluronic Acid 35 kDa on an In Vitro Model of Preterm Small Intestinal Injury and Healing Using Enteroid-Derived Monolayers
09:36

Effect of Hyaluronic Acid 35 kDa on an In Vitro Model of Preterm Small Intestinal Injury and Healing Using Enteroid-Derived Monolayers

Published on: July 28, 2022

1.8K

Area of Science:

  • Neurology
  • Infectious Diseases
  • Pediatrics

Background:

  • Infant botulism is a rare but serious neuroparalytic illness.
  • It is caused by the neurotoxin produced by Clostridium botulinum bacteria.

Observation:

  • A case study details the clinical progression and epidemiological investigation of infant botulism in a 3-month-old.
  • Initial symptoms included constipation, poor feeding, hypotonia, drooling, irritability, weak cry, and cranial nerve palsies.

Findings:

  • Early recognition of clinical signs is crucial for timely diagnosis.
  • The investigation highlighted the significant risk of honey as a source of Clostridium botulinum spores.

Implications:

  • Increased awareness can lead to faster diagnosis and appropriate management of infant botulism.
  • Honey should be strictly avoided for infants under 12 months to prevent exposure to Clostridium botulinum.