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Published on: May 24, 2016
Acute myocardial infarction in a child with myocardial bridge
Xiao-Dong Liu1, Chun-Lei Sun1, Su-Ping Mu1
1Department of Pediatrics, Weifang People's Hospital Affiliated to Weifang Medical College, Weifang 261041, China.
Insights
Myocardial bridge (MB) can cause myocardial infarction (MI) in children, a rare condition typically linked to Kawasaki disease. This case highlights MB as a potential pediatric MI cause.
Area of Science:
- Pediatric Cardiology
- Cardiovascular Pathology
Background:
- Myocardial infarction (MI) is uncommon in children.
- Kawasaki disease is the leading cause of pediatric MI.
- Myocardial bridges (MB) are congenital anomalies where a segment of coronary artery travels through heart muscle.
Observation:
- A 7.5-year-old boy presented with symptoms of acute myocardial infarction.
- Diagnostic evaluations included ECG, coronary CT angiography, and emission computed tomography.
- Differential diagnoses included MI, fulminant myocarditis, and coronary myocardial bridge.
Findings:
- The patient received treatment including thrombolysis, supportive care, and anti-inflammatory agents.
- Biomarkers (myocardial enzymes, troponin-I) normalized within 21 days.
- Electrocardiogram abnormalities persisted, indicating myocardial damage.
Implications:
- Myocardial bridging should be considered in the differential diagnosis of pediatric myocardial infarction.
- This case expands the understanding of rare causes of MI in pediatric populations.
- Further research is needed to elucidate the pathophysiology and management of MB-induced MI in children.
Background:
Myocardial infarction (MI) is rare in children, and Kawasaki disease is now recognized as the main cause for MI. In this report, we present a child with MI caused by myocardial bridge (MB).
Methods:
A 7.5-year-old boy was admitted to Weifang People's Hospital on September 16, 2008 for heart disease. By electrocardiogram, coronary CT angiography, emission computed tomography, and other examinations, he was initially diagnosed as having (1) acute inferior myocardial infarction and extensive anterior myocardial infarction; (2) fulminant myocarditis; or (3) coronary myocardial bridge. He was treated with oxygen, thrombolysis, myocardial nutrition, vitamin C (4.0 g per time), dexamethasone (7.5 mg per time), a large dose of gamma globulin, and interferon.
Results:
Myocardial enzymes, liver function, C-reactive protein, and troponin-I returned to normal at 21 days after treatment. At 29 days, electrocardiogram indicated that II, III, aVF, V4 - V6 leads had abnormal Q wave, and ST-T changed. The patient was discharged.
Conclusion:
Myocardial bridge may be one of the causes of MI in children.
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