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Related Concept Videos

Cerebral Hemispheres01:05

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The human brain, a complex organ, is functionally divided into two cerebral hemispheres—left and right. These hemispheres are interconnected by a structure of paramount importance, the corpus callosum. This substantial bundle of neural fibers is not just a bridge between the hemispheres but a crucial element for the brain's comprehensive functioning. It enables efficient communication between the two hemispheres, allowing each side of the brain to control and receive sensory and motor...
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Related Experiment Video

Updated: Apr 22, 2026

How to Create and Use Binocular Rivalry
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Published on: November 10, 2010

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Unihemispheric burst suppression.

Edward C Mader1, Nicole R Villemarette-Pittman1, Cornel T Rogers1

  • 1Epilepsy Center of Excellence, Louisiana State University Health Sciences Center , New Orleans, LA, USA.

Neurology International
|October 14, 2014
PubMed
Summary
This summary is machine-generated.

Unihemispheric burst suppression (BS) in EEG, unlike typical bilateral BS, may indicate an epileptic mechanism. This rare phenomenon resolved with propofol in two patients, suggesting an underlying seizure disorder.

Keywords:
burst suppressionelectroencephalogrampropofolseizurestatus epilepticusunihemispheric

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Area of Science:

  • Neuroscience
  • Clinical Neurology
  • Epileptology

Background:

  • Burst suppression (BS) is characterized by alternating high-voltage bursts and suppressed EEG activity.
  • Bihemispheric BS, induced by anesthesia or encephalopathy, is generally considered non-epileptic.
  • Unihemispheric BS is a rare variant with poorly understood clinical significance.

Observation:

  • Two cases of unihemispheric BS are presented in patients experiencing convulsive status epilepticus.
  • One patient had a left temporoparietal tumor; the other had complex medical issues and a history of seizures.
  • Both patients received propofol for seizure management, leading to the resolution of unihemispheric BS.

Findings:

  • Unihemispheric BS was observed in the left hemisphere in both cases.
  • Increasing propofol infusion rates abolished unihemispheric BS and improved clinical status.
  • The association with clinical seizures and resolution with propofol suggests an epileptic origin.

Implications:

  • The assumption that BS is non-epileptic should not be automatically applied to unihemispheric patterns.
  • Unihemispheric BS may serve as an indicator of underlying epileptic activity.
  • Further research is needed to elucidate the mechanisms and clinical implications of unihemispheric BS.