Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Arboviral Encephalitis01:25

Arboviral Encephalitis

52
Arboviral encephalitis refers to brain inflammation caused by arthropod-borne viruses, particularly those transmitted through mosquito vectors. Among these, West Nile virus (WNV), a member of the Flaviviridae family, is a significant public health concern. WNV is an enveloped, positive-sense, single-stranded RNA virus. Human infection typically begins when an infected mosquito introduces the virus into the dermis during feeding. The primary transmission cycle involves birds as amplifying hosts...
52
Viral Meningitis01:18

Viral Meningitis

165
Viral meningitis is the most common form of meningitis and is often referred to as aseptic meningitis to indicate the absence of bacterial involvement. It is generally milder than bacterial meningitis, with symptoms including fever, headache, stiff neck, drowsiness, nausea, photophobia, and vomiting. Rarely, more severe manifestations or death may occur. Common causative agents include enteroviruses, particularly coxsackie A and B viruses and echoviruses, all members of the Enterovirus genus...
165
Viral Hepatitis I: Introduction01:28

Viral Hepatitis I: Introduction

15
Viral hepatitis is an inflammatory condition of the liver caused by infection with hepatotropic viruses, most commonly hepatitis A, B, C, D, and E. Despite variations in structure and transmission, all viruses mentioned infect hepatocytes and provoke immune responses that can hinder liver function. Additionally, some non-hepatotropic viruses can also lead to hepatic inflammation.Hepatitis A VirusHepatitis A virus (HAV) is transmitted through the fecal–oral route, typically by ingestion...
15
Encephalitis l: Introduction01:19

Encephalitis l: Introduction

5
Encephalitis is inflammation of the brain parenchyma, most often due to infections or autoimmune processes. It presents with neuropsychiatric features such as fever, altered mental status, behavioral changes, cognitive dysfunction, seizures, focal deficits, and sometimes autonomic instability. In some cases, the meninges are also involved, resulting in meningoencephalitis.Infectious CausesInfectious encephalitis is most commonly viral but can also result from bacterial, fungal, or parasitic...
5
Encephalitis ll: Pathophysiology01:26

Encephalitis ll: Pathophysiology

9
Encephalitis is inflammation of the brain parenchyma caused by direct viral invasion or immune-mediated mechanisms triggered by infections or tumors. Both processes lead to neuronal injury, disrupted neurotransmission, and diverse neurological symptoms, often with overlapping clinical and pathological features.Autoimmune EncephalitisIn autoimmune encephalitis, antibodies target neuronal antigens on cell surfaces, synapses, or within neurons. A key example is anti-NMDAR encephalitis, which can...
9
Cytomegalovirus Disease01:27

Cytomegalovirus Disease

73
Cytomegalovirus (CMV) disease is caused by human cytomegalovirus, a double-stranded DNA virus of the Herpesviridae family. While primary CMV infection is often asymptomatic in immunocompetent individuals, the virus can cause severe disease in neonates and immunocompromised patients. CMV is the most common cause of congenital viral infection in the United States, and a major pathogen in solid organ and hematopoietic stem cell transplant recipients.CMV is transmitted via bodily fluids, sexual...
73

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

Reverse genetics approach for arteriviruses using circular polymerase extension reaction.

Access microbiology·2026
Same author

Pigs lacking Natural Killer T cells have altered cellular responses to influenza.

PLoS pathogens·2026
Same author

Influenza-Infected Pigs Are Not Susceptible to SARS-CoV-2 Infection.

Pathogens (Basel, Switzerland)·2026
Same author

Cell Surface Vimentin Is an Attachment Factor That Facilitates Equine Arteritis Virus Infection In Vitro.

Viruses·2026
Same author

Pigs lacking Natural Killer T cells have altered cellular responses to influenza.

bioRxiv : the preprint server for biology·2025
Same author

Coding-complete genome sequences of group B equine rotavirus from central Kentucky, USA, reveal circulation of a single genome constellation.

Microbiology resource announcements·2025

Related Experiment Video

Updated: Apr 20, 2026

Viral Concentration Determination Through Plaque Assays: Using Traditional and Novel Overlay Systems
09:28

Viral Concentration Determination Through Plaque Assays: Using Traditional and Novel Overlay Systems

Published on: November 4, 2014

117.8K

Equine viral arteritis.

Udeni B R Balasuriya1

  • 1Department of Veterinary Science, Maxwell H. Gluck Equine Research Center, College of Agriculture, Food and Environment, University of Kentucky, Lexington, KY 40546-0099, USA.

The Veterinary Clinics of North America. Equine Practice
|December 3, 2014
PubMed
Summary

Equine arteritis virus (EAV) causes equine viral arteritis (EVA), a global disease impacting horses and related species. This review covers EVA

Area of Science:

  • Veterinary Virology
  • Equine Infectious Diseases

Background:

  • Equine arteritis virus (EAV) causes equine viral arteritis (EVA), a significant global disease.
  • EVA affects equids (horses, donkeys, mules, zebras), causing respiratory and reproductive issues.
  • Outbreaks lead to substantial economic losses in the equine industry.

Purpose of the Study:

  • To provide a comprehensive summary of current knowledge on equine viral arteritis (EVA).
  • To detail the disease, its pathogenesis, and epidemiology.
  • To review host immune responses, vaccination, treatment, and control strategies for EVA.

Main Methods:

  • Literature review of scientific publications on EAV and EVA.
  • Synthesis of information on disease characteristics, transmission, and host interactions.
Keywords:
EAVEVAEquine arteritis virusEquine viral arteritis

More Related Videos

In Vivo Imaging Systems IVIS Detection of a Neuro-Invasive Encephalitic Virus
10:21

In Vivo Imaging Systems IVIS Detection of a Neuro-Invasive Encephalitic Virus

Published on: December 2, 2012

21.5K
Subconjunctival Administration of Adeno-associated Virus Vectors in Small Animal Models
06:16

Subconjunctival Administration of Adeno-associated Virus Vectors in Small Animal Models

Published on: March 16, 2022

3.8K

Related Experiment Videos

Last Updated: Apr 20, 2026

Viral Concentration Determination Through Plaque Assays: Using Traditional and Novel Overlay Systems
09:28

Viral Concentration Determination Through Plaque Assays: Using Traditional and Novel Overlay Systems

Published on: November 4, 2014

117.8K
In Vivo Imaging Systems IVIS Detection of a Neuro-Invasive Encephalitic Virus
10:21

In Vivo Imaging Systems IVIS Detection of a Neuro-Invasive Encephalitic Virus

Published on: December 2, 2012

21.5K
Subconjunctival Administration of Adeno-associated Virus Vectors in Small Animal Models
06:16

Subconjunctival Administration of Adeno-associated Virus Vectors in Small Animal Models

Published on: March 16, 2022

3.8K
  • Compilation of data on diagnostic, therapeutic, and preventive measures.
  • Main Results:

    • EAV is specific to the Equidae family.
    • EVA presents as a complex disease with varied clinical signs.
    • Current understanding encompasses pathogenesis, immune response, and control strategies.

    Conclusions:

    • EVA remains a critical concern for equine health and industry.
    • Effective management requires integrated approaches including vaccination and biosecurity.
    • Further research is needed to address remaining knowledge gaps and improve control.