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In-vitro effects of cilofungin (LY121019), amphotericin B and amphotericin B-deoxycholate on human polymorphonuclear

P Van der Auwera1, F Meunier

  • 1Service de Médecine Interne, Institut Jules Bordet, Bruxelles, Belgium.

Insights

Cilofungin and amphotericin B affect human immune cells (PMNs) differently. Cilofungin enhances phagocytosis of bacteria and fungi, while amphotericin B impairs immune cell adherence and superoxide generation.

Area of Science:

  • Immunology
  • Pharmacology
  • Mycology

Background:

  • Human polymorphonuclear leucocytes (PMNs) are crucial for innate immunity.
  • Antifungal agents like cilofungin and amphotericin B can modulate immune cell function.
  • Understanding these interactions is vital for optimizing antifungal therapy.

Purpose of the Study:

  • To investigate the in vitro effects of cilofungin and amphotericin B on human PMN functions.
  • To compare the impact of these antifungals on PMN adherence, ingestion, killing, and oxidative burst.
  • To assess these effects against various fungal and bacterial pathogens.

Main Methods:

  • Multifunctional in vitro assessment of human PMNs.
  • Exposure of PMNs to cilofungin and amphotericin B at various concentrations and time points (PRE, PER, POST).
  • Evaluation of PMN adherence, Staphylococcus aureus and Candida albicans ingestion, intracellular killing, MTT reduction, and superoxide generation.

Main Results:

  • High-concentration cilofungin enhanced PMN adherence and ingestion of S. aureus and C. albicans.
  • Amphotericin B and its deoxycholate formulation reduced PMN adherence and superoxide generation.
  • Both antifungals increased killing of yeasts when added before or during ingestion; amphotericin B-deoxycholate also increased killing when added after ingestion.

Conclusions:

  • Cilofungin demonstrates immunomodulatory effects, potentially enhancing PMN antifungal activity.
  • Amphotericin B exhibits immunosuppressive properties by reducing PMN adherence and oxidative burst.
  • The timing of antifungal administration significantly influences their impact on PMN-mediated host defense.

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