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Late Cardiac Tamponade as a Result of Parietal Pericardium Erosion
Stanislaw Tubek1, Piotr Niewinski1, Dariusz Jagielski2
1Department of Cardiology, 4(th) Military Hospital, Wroclaw, Poland; Department of Heart Disease, Faculty of Health Sciences, Medical University in Wroclaw, Wroclaw, Poland.
Insights
Late cardiac tamponade after implantable cardioverter-defibrillator (ICD) lead placement is rare. A unique case revealed a pressure sore from the ICD lead causing fatal cardiogenic shock, highlighting the need for awareness.
Area of Science:
- Cardiology
- Medical Devices
- Surgical Complications
Background:
- Late-onset cardiac tamponade is a rare complication following intracardiac lead implantation.
- Implantable cardioverter-defibritors (ICDs) are crucial for managing cardiac arrhythmias.
- Diagnostic and management challenges exist for delayed complications of cardiac device leads.
Observation:
- A case of cardiac tamponade leading to cardiogenic shock occurred 1,164 days post-ICD implantation.
- Surgical exploration revealed an uncommonly reported mechanism: a pressure sore caused by the ICD lead on the parietal pericardium.
- No visible damage to the visceral pericardium was noted.
Findings:
- The pressure sore on the parietal pericardium was the direct cause of the tamponade.
- This specific lead-induced pericardial erosion is a novel finding.
- The delayed presentation underscores the potential for late-stage device-related issues.
Implications:
- Prompt diagnosis and surgical intervention are critical for managing this rare complication.
- Physicians must consider lead-induced pericardial erosion as a potential cause of late cardiac tamponade.
- Increased awareness of this pathomechanism can prevent fatal outcomes associated with conservative management.
Abstract:
Late onset cardiac tamponade is a rare and particularly challenging (both from diagnostic and management perspectives) complication of intracardiac lead implantation. We present a case of a late tamponade leading to cardiogenic shock, which occurred 1,164 days after implantable cardioverter-defibrillator (ICD) implantation. Open repair revealed unusual and, to our knowledge, not yet reported mechanism of the disease. A pressure sore caused by an ICD lead was found in the parietal layer of pericardium with no visible damage to the visceral layer. Conservative management in the described clinical scenario could be fatal, thus awareness of this pathomechanism of tamponade is critical.
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