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Introduction and spread of multi-resistant Shigella dysenteriae I in Thailand

D N Taylor1, L Bodhidatta, J E Brown

  • 1Department of Bacteriology, Armed Forces Research Institute of Medical Sciences, Bangkok, Thailand.

Insights

Shigella dysenteriae I outbreaks in Thailand showed a drug-resistant strain. High infection rates occurred across all ages, highlighting the importance of susceptible populations in disease spread.

Area of Science:

  • Epidemiology
  • Infectious Diseases
  • Microbiology

Background:

  • Recurrent outbreaks of Shigella dysenteriae serotype I (S. dysenteriae I) occurred in northeastern Thailand in 1986 and 1987, marking the first emergence in over two decades.
  • The prevalent epidemic strain exhibited resistance to multiple antibiotics, including tetracycline, streptomycin, chloramphenicol, and trimethoprim-sulfamethoxazole, while remaining susceptible to ampicillin.

Purpose of the Study:

  • To investigate the epidemiology and characteristics of the S. dysenteriae I outbreaks in northeastern Thailand.
  • To determine the antibiotic resistance patterns and genetic basis of trimethoprim resistance in the epidemic strain.
  • To assess the clinical impact, including incidence, hospitalization, and case-fatality rates, and identify factors contributing to the rapid spread.

Main Methods:

  • Surveillance data collection on dysentery cases in Ubon Province.
  • Antimicrobial susceptibility testing of the S. dysenteriae I epidemic strain.
  • Molecular characterization of trimethoprim resistance, identifying type I dihydrofolate reductase.
  • Serological surveys using IgG antibody to S. dysenteriae I lipopolysaccharide to estimate infection prevalence.

Main Results:

  • In Ubon Province, 10,000 dysentery cases were reported, with an incidence of 3-5 per 1,000 residents during peak months and a 0.9% overall case-fatality rate among reported cases.
  • The epidemic S. dysenteriae I strain was resistant to tetracycline, streptomycin, chloramphenicol, and trimethoprim-sulfamethoxazole, with trimethoprim resistance encoded by type I dihydrofolate reductase.
  • Infections affected all age groups, unlike S. flexneri, and a village study indicated high attack rates (46%) and infection prevalence (76% estimated by serology).

Conclusions:

  • The re-emergence of S. dysenteriae I in Thailand was driven by a multi-drug resistant strain.
  • The susceptibility of the broader population across all age groups facilitated the rapid dissemination of the pathogen.
  • Effective control strategies must consider the antibiotic resistance profile and the widespread susceptibility within the population.

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