Charcot Foot

H Zwipp1, S Rammelt1, C Dahlen1

  • 1Klinik und Poliklinik für Unfall- und Wiederherstellungschirurgie, Universitätsklinikum Carl Gustav Carus, TU Dresden, Germany.

Der Orthopade
|March 2, 2017
PubMed

Insights

Charcot foot, a stage of hereditary motor and sensory neuropathy (HMSN), involves progressive muscle atrophy and severe foot/ankle deformities. Early detection and surgical interventions can prevent further complications and improve long-term outcomes.

Area of Science:

  • Neurology
  • Orthopedic Surgery
  • Genetics

Background:

  • Charcot foot is historically linked to stage 4 of hereditary motor and sensory neuropathy (HMSN), also known as Charcot-Marie-Tooth disease.
  • This progressive neurological disorder primarily affects the feet and ankles, leading to muscle weakness, deformities like pes cavus, and eventual arthropathy.
  • The condition can advance to stage 5, characterized by neuropathic fractures of the ankle due to repetitive microtrauma.

Purpose of the Study:

  • To differentiate Charcot foot in HMSN from diabetic Charcot ankle.
  • To outline diagnostic and therapeutic strategies for both conditions.
  • To emphasize the importance of early detection and appropriate management to prevent severe deformities and functional loss.

Main Methods:

  • Review of clinical presentations and historical definitions of Charcot foot.
  • Discussion of diagnostic tools, including nerve conduction studies.
  • Analysis of surgical interventions such as tendon transfers, osteotomies, arthrodesis, and amputation.
  • Emphasis on post-operative care including non-weight-bearing and immobilization.

Main Results:

  • Charcot foot in HMSN presents with intrinsic foot muscle weakness, progressing to fixed deformities and ankle arthrosis.
  • Diabetic arthropathy (Type IV) requires specific management, often tibiocalcanear arthrodesis.
  • Surgical interventions can correct deformities and prevent the need for more extensive procedures like triple arthrodesis.
  • Appropriate post-operative care significantly reduces implant failure rates.

Conclusions:

  • Early diagnosis of Charcot foot in HMSN through nerve conduction studies is crucial for timely intervention.
  • Surgical correction, including ankle or double arthrodesis, is indicated for advanced stages of Charcot foot and diabetic arthropathy.
  • Prolonged non-weight-bearing and immobilization are essential for successful surgical outcomes in both conditions.
  • Salvage procedures like modified Pirogoff amputation may be necessary for infection or necrosis.

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