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Surgery for Chronic Traumatic Atlantoaxial Dislocation Associated With Myelopathy
Lingjun Wang1, Yong Gu, Liang Chen
1Department of Orthopaedic Surgery, the First Affiliated Hospital of Soochow University, Suzhou, China.
Delayed myelopathy from atlantoaxial dislocation (AAD) poses risks. Surgical decompression and fusion are crucial for chronic AAD, with high fusion rates and improved outcomes, regardless of reduction.
Area of Science:
- Neurosurgery
- Orthopedic Surgery
- Spinal Cord Disorders
Background:
- Delayed myelopathy in atlantoaxial dislocation (AAD) is poorly understood.
- Optimal surgical timing and techniques for chronic AAD remain debated.
Purpose of the Study:
- To evaluate surgical outcomes for myelopathy secondary to atlantoaxial dislocation (AAD).
- To assess the efficacy of decompression and fusion in chronic AAD patients.
Main Methods:
- Retrospective study of 18 patients undergoing decompression, fixation, and fusion for AAD.
- Evaluation of demographic data, operative details, complications, and functional scores (JOA, VAS).
- Assessment of radiological parameters including fusion rate, space available for the cord, and compression indices.
Main Results:
- Surgical decompression and fusion led to improved neurological status in most patients.
- Significant improvements observed in Japanese Orthopedic Association scores and reduced visual analogue scale scores.
- High fusion rate (83%) and successful cord decompression (65.9%) achieved with favorable radiological changes.
Conclusions:
- Timely surgical intervention is critical to mitigate the risk of delayed myelopathy in AAD.
- Sufficient decompression and solid fusion are essential for chronic AAD, irrespective of complete reduction.
- Both C1-C2 and occipital-cervical fusion techniques yield satisfactory outcomes for reducible and irreducible AAD, respectively.
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