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Angioedema.

J Farnam, J A Grant

    Dermatologic Clinics
    |January 1, 1985
    PubMed
    Summary
    This summary is machine-generated.

    Angioedema involves swelling due to various mechanisms, including allergic reactions and kinin system activation. Hereditary angioedema, caused by C1INH deficiency, can be managed with androgens.

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    Area of Science:

    • Immunology
    • Allergy and Immunology
    • Genetics

    Background:

    • Angioedema presents as localized swelling affecting skin, oropharyngeal, or gastrointestinal tissues.
    • It arises from diverse mechanisms including IgE-mediated responses, complement activation, and kinin system activation.
    • Triggers range from foods and drugs to infections and hereditary factors, leading to acute or chronic presentations.

    Purpose of the Study:

    • To summarize the mechanisms, triggers, and management of angioedema.
    • To differentiate between acute and chronic angioedema and their diagnostic approaches.
    • To highlight the specific characteristics and diagnosis of hereditary angioedema.

    Main Methods:

    • Review of underlying pathophysiological mechanisms of angioedema.

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  • Categorization of angioedema into acute and chronic forms.
  • Description of diagnostic criteria and therapeutic strategies, including for hereditary angioedema.

    • Main Results:

    • Angioedema results from multiple pathways, including mast cell degranulation and kinin generation.
    • Acute angioedema is typically self-limiting, while chronic forms require thorough investigation.
    • Hereditary angioedema is linked to C1INH deficiency, with characteristic symptoms and diagnostic markers.

    Conclusions:

    • Angioedema management varies based on type and cause, with H1 antihistamines as a primary treatment.
    • Chronic angioedema necessitates exclusion of serious conditions.
    • Hereditary angioedema, a C1INH deficiency disorder, shows biochemical defects reversible by androgens.