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Published on: December 6, 2016
Pediatric Obstructive Sleep Apnea: Where Do We Stand?
Insights
Pediatric obstructive sleep apnea (OSA) presents differently in children, often with behavioral issues. Understanding its complex causes and exploring diverse treatments beyond adenotonsillectomy is crucial for effective management.
Area of Science:
- Pediatric Sleep Medicine
- Otolaryngology
- Developmental Pediatrics
Background:
- Pediatric obstructive sleep apnea (OSA) was first described in 1976 and distinguished from adult OSA in 1981.
- Childhood OSA is characterized by disturbed sleep and behavioral problems, including hyperactivity and psychiatric issues, differing from adult symptoms.
Purpose of the Study:
- To discuss the mechanisms of pediatric OSA.
- To investigate sleep-disordered breathing in obese and nonobese children.
- To review current and emerging treatments for pediatric OSA.
Main Methods:
- Review of existing literature on pediatric OSA.
- Analysis of factors contributing to upper airway size and dysfunction.
- Investigation of various treatment modalities including surgery, medication, orthodontics, positive airway pressure, and myofunctional therapy.
Main Results:
- Pediatric OSA has complex causes involving adenotonsillar hypertrophy, obesity, and anatomical factors.
- The concept of dysfunction leading to dysmorphism impacting upper airway size is a recent advancement.
- Adenotonsillectomy (T&A) is questioned as the sole treatment, prompting exploration of alternatives.
Conclusions:
- Understanding of pediatric OSA has advanced, with identified factors influencing its development.
- Current treatment options for pediatric OSA are diverse, including T&A, medication, orthodontic interventions, positive airway pressure, and myofunctional therapy.
- Pediatricians and specialists need greater awareness of recent advances and available remedies for pediatric OSA.
Abstract:
Pediatric obstructive sleep apnea (OSA) was initially described in 1976. In 1981, Dr. Guilleminault emphasized that pediatric OSA was different from the clinical presentation reported in adults. It was characterized by more disturbed nocturnal sleep than excessive daytime sleepiness, and presented more behavioral problems, particularly school problems, hyperactivity, nocturnal enuresis, sleep terrors, depression, insomnia, and psychiatric problems. The underlying causes of pediatric OSA are complex. Such factors as adenotonsillar hypertrophy, obesity, anatomical and neuromuscular factors, and hypotonic neuromuscular disease are also involved. Adenotonsillectomy (T&A) has been the recommended treatment for pediatric OSA, but in the recent past this practice has been placed very much in question. Therefore, we will discuss the mechanism of pediatric OSA and investigate obese and nonobese pediatric sleep-disordered breathing. Moreover, the important concept that dysfunction leads to the dysmorphism that impacts on the size of the upper airway has been advanced recently. Finally, the treatments of pediatric OSA, such as T&A, medication, the orthodontic approaches (rapid maxillary expansion, or mandibular advancement with functional appliances), positive airway pressure, and noninvasive treatment, such as myofunctional therapy (MFT), will be investigated. A "passive MFT" has been tried recently, but very few results exist. In conclusion, we have made progress in our understanding of pediatric OSA, and we can even recognize factors leading to its development or worsening. However, pediatricians and pediatric subspecialists are often unaware of the advances and the remedies available.
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