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Published on: December 28, 2014
Ataxic Hemiparesis Associated with Cortical Infarct Localized in the Postcentral Gyrus
Yoshino Kinjo1, Satoshi Suda1, Yuki Sakamoto1
1Department of Neurological Science, Graduate School of Medicine, Nippon Medical School, Japan.
Insights
Ataxic hemiparesis (AH), a lacunar syndrome, typically involves brain lesions and crossed cerebellar diaschisis (CCD). This case study presents an AH patient with postcentral gyrus lesions who did not exhibit CCD.
Area of Science:
- Neurology
- Neuroscience
- Cerebrovascular Diseases
Background:
- Ataxic hemiparesis (AH) is a lacunar syndrome classically linked to subcortical lesions.
- Crossed cerebellar diaschisis (CCD), a functional deficit in the cerebellum, often accompanies AH.
- Lesions in the postcentral gyrus are rarely associated with AH.
Observation:
- This report describes a patient experiencing AH due to an acute infarction.
- The infarction was localized to the postcentral gyrus, a cortical area.
- The patient did not present with crossed cerebellar diaschisis (CCD).
Findings:
- The case demonstrates that AH can result from localized cortical lesions.
- Absence of CCD was noted despite the presence of AH and cortical lesion.
- This challenges the typical association between AH and CCD.
Implications:
- The findings suggest that CCD may not be a universal feature of all AH cases.
- Cortical lesions, specifically in the postcentral gyrus, should be considered in AH diagnosis.
- Further research is needed to understand the mechanisms underlying AH without CCD.
Abstract:
Ataxic hemiparesis (AH) is a classic lacunar syndrome associated with localized damage to the pons, internal capsule, thalamus, or corona radiata. A depression of metabolic activity known as crossed cerebellar diaschisis (CCD) is frequently observed in the cerebellar hemisphere contralateral to the site of the lesion in patients with AH. Though small cortical or subcortical lesions may result in AH, such occurrences are rare. The current report details the case of a patient with AH resulting from acute infarction associated with localized lesions of the postcentral gyrus who presented without CCD.
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