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Updated: Jul 21, 2026

An Ivor Lewis Esophagectomy Designed to Minimize Anastomotic Complications and Optimize Conduit Function
Published on: April 17, 2020
[Complete and inverted esophagastric duplicity]
J M Morán Penco1, J Vázquez2, E Forsheden Ahs3
1Cátedra de Cirugía. Universidad de Extremadura. Clideba Quirón Salud. Badajoz.
Insights
A rare gastric duplication in an infant was surgically removed, along with an unusual esophageal tubular structure. This case suggests congenital digestive malformations may stem from early genetic errors.
Area of Science:
- Pediatric Surgery
- Gastroenterology
- Developmental Biology
Background:
- Gastric duplications are rare congenital anomalies.
- They can present with various symptoms depending on size and location.
- Associated anomalies are uncommon but significant.
Observation:
- A 4-month-old female presented with an abdominal mass.
- Surgical exploration revealed a gastric duplication and an unusual tubular structure.
- The tubular structure, histologically identified as esophagus, connected to the duplication and inserted into the diaphragm.
Findings:
- A novel association of gastric duplication with an esophageal tubular malformation was identified.
- Surgical excision of both anomalies was successfully performed.
- Histological examination confirmed the nature of the malformation.
Implications:
- This case represents a previously unreported type of digestive tract duplication.
- It supports the hypothesis that such malformations arise from genetic transcription errors during early embryonic development.
- Further research into the genetic basis of foregut malformations is warranted.
Case Report:
A 4-month old girl affected by gastric duplication discovered as a palpable abdominal mass displacing the spleen and left kidney. During laparotomy, a yellowish secretion is aspirated and the gastric duplication is excised together with the common muscular Wall of the greater curvature of the stomach. Also excised, a tubular structure starting from the inferior pole of the malformation, and communicating with it, passing superior to the head of the pancreas and the second portion of the duodenum, followed by a posterior course before inserting into the right cruz of the diaphragm. The histology of the tubular formation was compatible with esophagus.
Conclusion:
This type of duplication has not been reported in the literature and reinforces the theory that these malformations could be secondary to genetic transcription errors, in the differentiation of the endoderm of the primitive digestive tract and the notochord.
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