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Published on: July 7, 2013
Persistent Buttock Claudication after Endovascular Abdominal Aortic Aneurysm Repair
Alessandro Robaldo1, Stefano Pagliari1, Filippo Piaggio1
1Vascular and Endovascular Surgery Unit, Imperia Hospital, Imperia, Italy.
Insights
A patient with disabling buttock claudication after internal iliac artery (IIA) embolization underwent successful open surgical repair. This highlights open surgery as a viable option for preserving pelvic inflow in high-risk patients.
Area of Science:
- Vascular Surgery
- Endovascular Interventions
- Aortoiliac Disease
Background:
- Endovascular aneurysm repair (EVAR) is common for aortoiliac aneurysms.
- Internal iliac artery (IIA) embolization can cause buttock claudication.
- Pelvic inflow preservation is crucial for preventing ischemic complications.
Observation:
- A 71-year-old male experienced disabling right buttock claudication post-right IIA coil embolization.
- CT angiography showed extensive aortoiliac calcifications and thrombus.
- Symptoms persisted despite contralateral IIA patency and collateral flow.
Findings:
- The patient underwent successful open infrarenal abdominal aortic aneurysm repair with stent-graft removal.
- Concomitant revascularization of the right IIA was performed.
- The patient remained asymptomatic at 30-month follow-up.
Implications:
- Open surgery can be a successful solution for buttock claudication after IIA embolization.
- Preserving pelvic inflow is vital, especially in high-risk patients with complex atheroma.
- Consideration of open repair is important for managing complications of EVAR adjuncts.
Abstract:
We describe the successful surgical treatment of a 71-year-old man affected by right buttock claudication after a right internal iliac artery (IIA) coil embolization as an adjunct to endovascular iliac artery aneurysm repair. Computed tomography angiography revealed extensive aortoiliac calcifications and thrombus in the vessel walls. Despite patency of the contralateral IIA and preservation of right distal collateral flow through ipsilateral hypogastric branches, the symptom was persistent and disabling. The high-risk patient underwent an "open" repair of the infrarenal abdominal aneurysm with removal of the entire stent-graft and concomitant revascularization of the right IIA. Post-operative recovery was uneventful, and the patient remained asymptomatic during a 30-month follow-up. This case underscores the importance of considering all potential solutions, including open surgery, to preserve pelvic inflow after aortoiliac stent grafting, particularly for high-risk patients with vulnerable plaque and higher risk of thrombus embolization.
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