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Related Concept Videos

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Cardiac Output and Stroke Volume01:11

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Cardiac output (CO) is an integral aspect of human physiology, reflecting the heart's efficiency and responsiveness to the body's needs. It represents the volume of blood that the left or right ventricle ejects into the aorta or pulmonary trunk each minute. The CO is calculated by multiplying the heart rate (HR)—the number of heartbeats per minute—by the stroke volume (SV)—the amount of blood pumped out with each heartbeat.
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Cardiac output (CO), the amount of blood the heart pumps per minute, is a parameter in cardiovascular physiology determined by stroke volume and heart rate. Stroke volume, the amount of blood pushed from one of the ventricles per heartbeat, is influenced by preload, afterload, and contractility.
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To wash hands properly, follow these steps:
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Zhurnal nevrologii i psikhiatrii imeni S.S. Korsakova·2026
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Related Experiment Video

Updated: Feb 9, 2026

Utilizing Repetitive Transcranial Magnetic Stimulation to Improve Language Function in Stroke Patients with Chronic Non-fluent Aphasia
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Utilizing Repetitive Transcranial Magnetic Stimulation to Improve Language Function in Stroke Patients with Chronic Non-fluent Aphasia

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[The prognosis for post-stroke aphasia].

V V Alferova1, V M Shklovskij2, E G Ivanova1

  • 1Center of Speech Pathology and Neurorehabilitation, Moscow, Russia; Pirogov Russian National Research Medical University, Moscow, Russia.

Zhurnal Nevrologii I Psikhiatrii Imeni S.S. Korsakova
|June 5, 2018
PubMed
Summary
This summary is machine-generated.

Severe neurological symptoms and decreased daily activity predict poor post-stroke aphasia recovery. Higher education and longer duration positively influence speech function recovery in ischemic stroke patients.

Keywords:
aphasia regressionclinical form of aphasiaischemic strokeprognostic factorsseverity of aphasia

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Area of Science:

  • Neuroscience
  • Neurology
  • Speech-Language Pathology

Background:

  • Post-stroke aphasia significantly impacts communication and quality of life.
  • Early recovery period is crucial for assessing prognostic factors.
  • Understanding predictors of aphasia regression is vital for targeted rehabilitation.

Purpose of the Study:

  • To identify risk factors for poor aphasia regression.
  • To determine factors associated with favorable prognosis for aphasia recovery.
  • To investigate the influence of clinical, biological, and social factors on post-stroke aphasia.

Main Methods:

  • Cohort study of 40 patients with ischemic stroke and aphasia.
  • Assessment of speech and non-speech cognitive functions over 3-4 months.
  • Logistic regression and odds ratio estimation to analyze prognostic factors.

Main Results:

  • Higher neurological symptom severity (NIHSS) and lower daily activity (Barthel index) were associated with poorer aphasia regression.
  • MRI findings in specific brain regions impacted non-verbal cognition.
  • Patients with initially severe sensory and sensorimotor aphasias showed significant speech improvement.
  • Higher education level and longer education duration positively correlated with aphasia regression.

Conclusions:

  • Neurological deficits worsen functional reorganization of speech networks.
  • Initial aphasia severity and clinical form influence recovery dynamics.
  • Education level and duration are key factors promoting post-stroke neuroplasticity and recovery.