Related Experiment Video
Updated: Feb 7, 2026

05:47
Use of a Rat Model to Study Ventral Abdominal Hernia Repair
Published on: October 2, 2017
9.8K
Retromuscular Sublay Technique for Ventral Hernia Repair
Irfan A Rhemtulla1, John P Fischer1
1Division of Plastic Surgery, Department of Surgery, University of Pennsylvania, Philadelphia, Pennsylvania.
Seminars in Plastic Surgery
|July 27, 2018
Summary
The Rives-Stoppa sublay hernia repair technique, though old, offers significant benefits. Modern modifications enhance surgical outcomes and patient quality of life, making it a valuable approach.
Area of Science:
- General Surgery
- Surgical Innovation
Background:
- The sublay technique for hernia repair was introduced by Rives and Stoppa over 50 years ago.
- Its full clinical potential and benefits have been realized more recently due to modifications and increased appreciation.
Purpose of the Study:
- To clarify the nomenclature surrounding the sublay, retrorectus, retromuscular, and preperitoneal surgical planes.
- To explain the historical context, surgical technique, outcomes, and future considerations of the Rives-Stoppa procedure.
Main Methods:
- Literature review and analysis of surgical nomenclature.
- Description of the retromuscular approach and its anatomical considerations.
- Highlighting modifications and their impact on surgical outcomes.
Main Results:
- The retromuscular approach, a key component of the Rives-Stoppa technique, requires significant anatomical knowledge and surgical skill.
- Modifications have led to improved postsurgical outcomes and enhanced patient quality of life.
- Clarification of terminology is essential for understanding the nuances of the surgical planes.
Conclusions:
- The Rives-Stoppa sublay technique, despite its age, remains a crucial surgical approach for hernia repair.
- Ongoing modifications continue to refine the procedure, improving patient recovery and long-term results.
- Understanding the precise terminology and anatomical planes is vital for successful application and further development.
Related Concept Videos
Mismatch Repair
43.7K
Overview
43.7K
Mismatch Repair
6.6K
Organisms are capable of detecting and fixing nucleotide mismatches that occur during DNA replication. This sophisticated process requires identifying the new strand and replacing the erroneous bases with correct nucleotides. Mismatch repair is coordinated by many proteins in both prokaryotes and eukaryotes.
The Mutator Protein Family Plays a Key Role in DNA Mismatch Repair
The human genome has more than 3 billion base pairs of DNA per cell. Prior to cell division, that vast amount of genetic...
The Mutator Protein Family Plays a Key Role in DNA Mismatch Repair
The human genome has more than 3 billion base pairs of DNA per cell. Prior to cell division, that vast amount of genetic...
6.6K
Overview of DNA Repair
33.8K
In order to be passed through generations, genomic DNA must be undamaged and error-free. However, every day, DNA in a cell undergoes several thousand to a million damaging events by natural causes and external factors. Ionizing radiation such as UV rays, free radicals produced during cellular respiration, and hydrolytic damage from metabolic reactions can alter the structure of DNA. Damages caused include single-base alteration, base dimerization, chain breaks, and cross-linkage.
Chemically...
Chemically...
33.8K
Base Excision Repair
26.4K
One of the common DNA damages is the chemical alteration of single bases by alkylation, oxidation, or deamination. The altered bases cause mispairing and strand breakage during replication. This type of damage causes minimal change to the DNA double helix structure and can be repaired by the base excision repair (BER) pathways. BER corrects damaged DNA sequences by removing the damaged base and restoring the original base sequence using the complementary strand as a template.
The first step of...
The first step of...
26.4K
Nucleotide Excision Repair
40.9K
Overview
40.9K
Nucleotide Excision Repair
5.2K
DNA Distortion and Damage
Cells are regularly exposed to mutagens—factors in the environment that can damage DNA and generate mutations. UV radiation is one of the most common mutagens and is estimated to introduce a significant number of changes in DNA. These include bends or kinks in the structure, which can block DNA replication or transcription. If these errors are not fixed, the damage can cause mutations, which in turn can result in cancer or disease depending on which sequences are...
Cells are regularly exposed to mutagens—factors in the environment that can damage DNA and generate mutations. UV radiation is one of the most common mutagens and is estimated to introduce a significant number of changes in DNA. These include bends or kinks in the structure, which can block DNA replication or transcription. If these errors are not fixed, the damage can cause mutations, which in turn can result in cancer or disease depending on which sequences are...
5.2K

