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Related Experiment Video

Updated: Feb 6, 2026

A Novel Human Epithelial Enteroid Model of Necrotizing Enterocolitis

A Novel Human Epithelial Enteroid Model of Necrotizing Enterocolitis

Published on: April 10, 2019

Neurodevelopmental outcomes following necrotizing enterocolitis.

Marie Hickey¹, Michael Georgieff², Sara Ramel¹

¹Department of Pediatrics, Division of Neonatology, University of Minnesota, Minneapolis, MN, USA.

Seminars in Fetal & Neonatal Medicine

|August 27, 2018

Summary

Necrotizing enterocolitis (NEC) in premature infants increases the risk of poor neurodevelopmental outcomes. Early, multifaceted follow-up is crucial for detection and intervention to optimize brain development.

Keywords:

Gastrointestinal microbiota Inflammation Necrotizing enterocolitis Neurodevelopment Nutrition Outcomes Prematurity

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Last Updated: Feb 6, 2026

A Novel Human Epithelial Enteroid Model of Necrotizing Enterocolitis

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Published on: April 10, 2019

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Published on: June 8, 2022

A Neonatal BALB/c Mouse Model of Necrotizing Enterocolitis

A Neonatal BALB/c Mouse Model of Necrotizing Enterocolitis

Published on: November 30, 2021

Area of Science:

Neonatal Medicine
Developmental Pediatrics
Pediatric Gastroenterology

Background:

Necrotizing enterocolitis (NEC) is a critical gastrointestinal condition in premature infants.
NEC is linked to significant risks of adverse neurodevelopmental outcomes.
The infant brain is highly vulnerable during the period when NEC typically occurs.

Purpose of the Study:

To highlight the association between NEC and neurodevelopmental impairments.
To emphasize the need for comprehensive follow-up strategies for affected infants.
To underscore the importance of early detection and intervention for optimizing neurodevelopmental trajectories.

Main Methods:

Review of existing literature on NEC and neurodevelopmental outcomes.
Discussion of the multifactorial pathogenesis of neurodevelopmental impairment post-NEC.
Outline of recommended multifaceted follow-up protocols.

Main Results:

NEC significantly increases the risk of poor neurodevelopmental outcomes in premature infants.
Both nutritional and non-nutritional factors contribute to neurodevelopmental deficits after NEC.
A comprehensive follow-up approach is essential for managing these risks.

Conclusions:

Optimizing neurodevelopmental outcomes in infants with NEC requires a proactive and multifaceted follow-up strategy.
Continued research is vital to elucidate NEC's impact on brain development and to develop targeted interventions.
Early and serial assessments, including developmental, neurologic, and sensory evaluations, are critical.