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Myoclonus induced by salbutamol: A case report
María Alejandra Montoya-Giraldo1, Dayana Vanessa Montoya, David Alexander Atehortúa
1Centro de Información y Estudio de Medicamentos y Tóxicos, Departamento de Farmacología y Toxicología, Facultad de Medicina, Universidad de Antioquia, Medellín, Colombia. maria.montoya4@udea.edu.co.
Biomedica : Revista Del Instituto Nacional De Salud
|October 19, 2018
Summary
Salbutamol, a common asthma medication, can rarely cause myoclonus, an involuntary muscle twitching. Discontinuing salbutamol resolved the patient's severe myoclonus, highlighting a potential adverse effect.
Area of Science:
- Pharmacology
- Neurology
- Pulmonology
Background:
- Salbutamol is a widely used beta-2 adrenergic agonist for obstructive and restrictive lung diseases.
- Common side effects include tachycardia and tremor.
- Myoclonus is characterized by sudden, brief, involuntary muscle contractions.
Observation:
- A 61-year-old patient presented with treatment-resistant myoclonus.
- The myoclonus significantly improved only after discontinuing salbutamol.
- This case highlights a potential adverse drug reaction to salbutamol.
Findings:
- This report details a case of salbutamol-induced myoclonus, a rare adverse effect.
- The patient's myoclonus resolved upon cessation of the beta-2 adrenergic agonist.
- This is the fourth reported instance of salbutamol-induced myoclonus in medical literature.
Implications:
- Clinicians should consider salbutamol as a potential cause of myoclonus in patients with lung disease.
- Early identification and discontinuation of the causative agent are crucial for managing drug-induced myoclonus.
- Further research is warranted to understand the mechanism and prevalence of salbutamol-induced myoclonus.
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