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Updated: Jan 24, 2026

Induction of Endothelial Differentiation in Cardiac Progenitor Cells Under Low Serum Conditions

Induction of Endothelial Differentiation in Cardiac Progenitor Cells Under Low Serum Conditions

Published on: January 7, 2019

SERUM OMENTIN-1 LEVELS AND ENDOTHELIAL DYSFUNCTION IN OBESITY.

A R Çimen¹, E T Cerit¹, O T Iyidir¹

¹Gazi University Faculty of Medicine, Department of Endocrinology and Metabolism, Ankara, Turkey.

Acta Endocrinologica (Bucharest, Romania : 2005)

|June 1, 2019

Summary

This summary is machine-generated.

Serum omentin-1 levels are lower in obese patients, correlating with poorer endothelial function. These findings suggest omentin-1 may serve as an early indicator of endothelial dysfunction in obesity.

Keywords:

Endothelial Dysfunction Flow Mediated Dilatation Obesity Omentin-1

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Isolation of Endothelial Progenitor Cells from Healthy Volunteers and Their Migratory Potential Influenced by Serum Samples After Cardiac Surgery

Isolation of Endothelial Progenitor Cells from Healthy Volunteers and Their Migratory Potential Influenced by Serum Samples After Cardiac Surgery

Published on: February 14, 2017

Area of Science:

Endocrinology
Cardiovascular Research
Metabolic Syndrome

Background:

Obesity is associated with increased cardiovascular risk.
Endothelial dysfunction is an early predictor of atherosclerosis.
Omentin-1 is an adipokine with potential protective roles.

Purpose of the Study:

To investigate the association between serum omentin-1 levels and endothelial dysfunction in obese individuals.
To explore the relationship between omentin-1 and metabolic parameters in obesity.

Main Methods:

Cross-sectional study comparing 50 obese patients with 45 healthy controls.
Measurements included oral glucose tolerance test, lipid profile, uric acid, HOMA-IR, serum omentin-1, and flow-mediated dilation (FMD).
Body composition analyzed using bioelectrical impedance.

Main Results:

Obese patients exhibited significantly lower serum omentin-1 levels and reduced FMD compared to controls.
Serum omentin-1 showed a positive correlation with FMD (r=0.359, p<0.001).
Omentin-1 levels were negatively correlated with BMI, waist circumference, body fat percentage, visceral fat, fasting insulin, and HOMA-IR.

Conclusions:

Reduced serum omentin-1 levels and impaired FMD may indicate early endothelial dysfunction in obesity.
Omentin-1 warrants further investigation as a potential biomarker for cardiovascular risk in obese populations.