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Related Experiment Videos

Unilateral hyperhidrosis after cerebral infarction.

D R Labar1, J P Mohr, F T Nichols

  • 1Department of Neurology, Columbia-Presbyterian Medical Center, New York, NY.

Neurology
|November 1, 1988
PubMed
Summary
This summary is machine-generated.

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Acute cerebral infarction can cause temporary unilateral hyperhidrosis (excessive sweating) on the opposite side of the body. This sign, linked to stroke, suggests a poor prognosis and disrupted brain pathways controlling sweating.

Area of Science:

  • Neurology
  • Autonomic Nervous System Research
  • Stroke Medicine

Background:

  • Cerebral infarction, a type of stroke, can manifest with diverse neurological deficits.
  • Autonomic dysfunction following stroke is recognized but specific signs require further elucidation.

Observation:

  • Six patients presented with unilateral hyperhidrosis contralateral to acute cerebral infarctions.
  • Hyperhidrosis predominantly affected the face and arm, lasting 1-3 days.
  • No other concurrent autonomic dysfunctions were noted in these patients.

Findings:

  • Infarctions varied from localized opercular cortex lesions to large strokes affecting cortical and subcortical structures.
  • The contralateral hyperhidrosis suggests a disruption of inhibitory cortical pathways regulating sweating.

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  • The presence of this sign correlated with significant residual neurological disability and one mortality.

    • Implications:

    • Unilateral hyperhidrosis may serve as an indicator of stroke severity and prognosis.
    • Further research into central autonomic pathways affected by cerebral infarction is warranted.
    • Understanding this sign can aid in early stroke assessment and patient outcome prediction.