MDA-9/Syntenin (SDCBP): Novel gene and therapeutic target for cancer metastasis

Swadesh K Das1, Santanu Maji2, Stephen L Wechman2

  • 1Department of Human and Molecular Genetics, Virginia Commonwealth University, School of Medicine, Richmond, VA, USA; VCU Institute of Molecular Medicine, Virginia Commonwealth University, School of Medicine, Richmond, VA, USA; VCU Massey Cancer Center, Virginia Commonwealth University, School of Medicine, Richmond, VA, USA.

Pharmacological Research
|February 17, 2020
PubMed

Insights

Targeting the pro-metastatic gene MDA-9/Syntenin (SDCBP) offers a promising strategy to inhibit cancer metastasis. Inhibiting MDA-9 suppresses tumor spread and sensitizes cancer cells to other therapies.

Area of Science:

  • Oncology
  • Molecular Biology
  • Cancer Research

Background:

  • Metastasis is the primary cause of cancer-related death from solid tumors.
  • Current treatments for metastasis are limited, highlighting the need for novel therapeutic targets.
  • Understanding the genetic and epigenetic factors driving metastasis is crucial for developing effective interventions.

Purpose of the Study:

  • To provide an overview of the metastatic process.
  • To highlight melanoma differentiation associated gene-9/Syntenin (MDA-9/Syntenin), also known as syndecan binding protein (SDCBP), as a key pro-metastatic gene.
  • To discuss the therapeutic potential of targeting MDA-9/Syntenin in various cancers.

Main Methods:

  • Review of the metastatic cascade, including invasion, intravasation, angiogenesis, and extravasation.
  • Analysis of MDA-9/Syntenin expression patterns in different cancer types.
  • Evaluation of pre-clinical studies investigating the effects of MDA-9/Syntenin inhibition (genetic and pharmacological) on metastasis.

Main Results:

  • MDA-9/Syntenin is significantly elevated in a wide range of carcinomas and other cancers, including melanoma and neuroblastoma.
  • Inhibition of MDA-9/Syntenin genetically or pharmacologically profoundly suppresses metastatic spread.
  • Blocking MDA-9/Syntenin enhances the efficacy of other therapeutic agents, converting anti-invasion effects into tumor cell death.

Conclusions:

  • MDA-9/Syntenin is a critical driver of metastasis across multiple cancer types.
  • Targeting MDA-9/Syntenin presents a viable therapeutic strategy to combat cancer metastasis.
  • Combined therapeutic approaches involving MDA-9/Syntenin inhibition show promise for improved patient outcomes.

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