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Related Concept Videos

Chronic Pancreatitis I: Introduction01:24

Chronic Pancreatitis I: Introduction

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The pancreas, an elongated and flat gland situated behind the stomach, serves a vital function in digesting food and managing blood sugar levels.
Pancreatitis is the inflammation of the pancreas, which occurs when the immune system becomes active and causes swelling, pain, and disruptions in organ function. Pancreatitis can manifest as either an acute or chronic condition.
Acute pancreatitis arises suddenly and lasts for a brief duration, while chronic pancreatitis is a long-term affliction...
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Acute Pancreatitis I: Introduction01:27

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Pancreatitis is inflammation of the pancreas, an organ located behind the stomach. It can be either acute or chronic.
Acute pancreatitis is characterized by rapid inflammation of the pancreas, often caused by factors like gallstone blockage or excessive alcohol consumption. Chronic pancreatitis, on the other hand, is a slow, progressive inflammation that may result from long-term alcohol abuse, obstructions in the pancreatic duct, or genetic factors.
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Acute Pancreatitis II: Clinical Manifestations and Management01:30

Acute Pancreatitis II: Clinical Manifestations and Management

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Acute pancreatitis presents a complex medical emergency characterized by rapid onset inflammation of the pancreas, demanding timely diagnosis and management to prevent complications. The condition primarily manifests through severe upper abdominal pain that often radiates to the back. This pain intensifies following the consumption of fatty foods. Accompanying symptoms such as nausea, vomiting, abdominal distention, fever, dyspnea, cyanosis, and jaundice can vary in intensity but significantly...
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The Parathyroid Glands00:59

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The two pairs of parathyroid glands embedded within the posterior surface of the thyroid gland are restricted by a dense capsule around them. These glands comprise two distinct cell populations—parathyroid oxyphil and parathyroid principal cells- pivotal in calcium homeostasis.
Oxyphil cells, whose functions remain elusive, emerge during late puberty, adding a layer of complexity to the parathyroid gland's intricacies. In contrast, principal parathyroid cells undertake a vital role by...
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Chronic Pancreatitis II: Collaborative Care01:29

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The management of chronic pancreatitis is multifaceted, involving a comprehensive approach that includes thorough assessment, diagnostic testing, and a variety of management strategies.
Assessment:
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Gastritis-II: Pathophysiology01:17

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Gastritis is marked by disruption of the mucosal barrier that usually protects the stomach tissue from digestive juices and manifests in acute and chronic forms.
In acute gastritis, the gastric mucosa becomes swollen and red and undergoes superficial erosion. Superficial ulceration may lead to bleeding.
In chronic gastritis, persistent or repeated insults lead to chronic inflammatory changes and, eventually, thinning or atrophy of the gastric tissue.
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Establishment of a Simple and Effective Rat Model for Intraoperative Parathyroid Gland Imaging
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Primary hyperparathyroidism and pancreatitis.

R A Misgar1, M H Bhat2, T A Rather3

  • 1Department of Endocrinology, Sher-i-Kashmir Institute of Medical Sciences, Srinagar, Kashmir, India. drreyaz07@rediffmail.com.

Journal of Endocrinological Investigation
|April 8, 2020
PubMed
Summary
This summary is machine-generated.

Primary hyperparathyroidism (PHPT) can present solely with pancreatitis. Investigating PHPT in pancreatitis patients with elevated calcium is crucial, as parathyroidectomy often resolves pancreatitis symptoms.

Keywords:
HypercalcemiaPancreatitisPrimary hyperparathyroidism

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Area of Science:

  • Endocrinology
  • Gastroenterology
  • Surgical Research

Background:

  • The association between primary hyperparathyroidism (PHPT) and pancreatitis remains debated.
  • Clinical data on PHPT patients experiencing pancreatitis are limited.

Purpose of the Study:

  • To present clinical data, investigations, management, and follow-up of PHPT patients with pancreatitis.
  • To compare PHPT patients with pancreatitis against those without.

Main Methods:

  • Retrospective analysis of 242 PHPT patients over 24 years.
  • Pancreatitis diagnosis based on abdominal pain, elevated amylase (>3x normal), or imaging findings.
  • Demographic and laboratory data were collected.

Main Results:

  • 6.19% of PHPT patients (15/242) developed pancreatitis, predominantly acute (14/15).
  • Pancreatitis was the initial symptom in 93.3% of affected patients, with no other risk factors identified.
  • PHPT patients with pancreatitis showed higher serum calcium and alkaline phosphatase (ALP) levels.

Conclusions:

  • Pancreatitis can be the sole presenting symptom of PHPT.
  • Investigating PHPT is vital for pancreatitis patients with high-normal or elevated calcium, especially when other causes are absent.
  • Parathyroidectomy led to pancreatitis resolution in most cases.