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Stereotaxic microinjection of HSV-1 selectively decreases striatal dopamine concentrations in mice

R F Seegal1, D J McFarland

  • 1Wadsworth Center for Laboratories and Research, New York State Department of Health, Albany 12201.

Brain Research
|April 5, 1988
PubMed

Insights

Herpes simplex virus type 1 infection in mice reduced dopamine levels in the striatum. This viral effect on dopamine metabolism occurred without significant cell damage or infection of dopaminergic neurons.

Area of Science:

  • Neuroscience
  • Virology
  • Biochemistry

Background:

  • Herpes simplex virus (HSV) can cause neurological complications.
  • The impact of HSV on neurotransmitter systems, particularly dopamine, is not fully understood.

Purpose of the Study:

  • To investigate the effect of HSV type 1 infection on dopamine metabolism in the mouse striatum.
  • To determine if HSV infection alters norepinephrine and serotonin levels.

Main Methods:

  • BALB/c mice were stereotaxically injected with HSV-1 (MP or MacIntyre strain) in the striatum.
  • Neurotransmitter and metabolite concentrations (dopamine, norepinephrine, serotonin) were measured using high-performance liquid chromatography with electrochemical detection.
  • Immunoperoxidase staining was used to assess viral spread and neuronal damage.

Main Results:

  • HSV-1 infection led to reduced dopamine levels and an elevated homovanillic acid to dopamine ratio in the striatum.
  • Norepinephrine and serotonin levels, along with their metabolites, remained unaffected by the infection.
  • Viral infection was confined to the striatum with minimal necrosis observed at three days post-infection.

Conclusions:

  • Herpes simplex virus type 1 can alter dopamine metabolism in the striatum.
  • These metabolic changes occur early in infection, preceding significant neuronal damage or detectable infection of dopaminergic neuron cell bodies.

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