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Relationship between atherogenic indices and acquired premature ejaculation.

Kemal Ertas1, Cemal Nas2

  • 1Department of Urology, Memorial Hospital, Diyarbakir, Turkey.

Andrologia
|December 8, 2020
PubMed
Summary

Acquired premature ejaculation (PE) is linked to higher triglyceride and Atherogenic Index of Plasma (AIP) levels. Increased AIP correlates with PE severity, suggesting a connection between lipid profiles and premature ejaculation.

Keywords:
acquiredatherogenic indexcastelli indexdyslipidemiapremature ejaculation

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Area of Science:

  • Urology
  • Cardiovascular Health
  • Endocrinology

Background:

  • Premature ejaculation (PE) is a common sexual dysfunction.
  • Blood lipid parameters and atherogenic indices are indicators of cardiovascular risk.
  • The relationship between PE and metabolic factors requires further investigation.

Purpose of the Study:

  • To evaluate blood lipid profiles and atherogenic indices in men with acquired premature ejaculation (PE).
  • To determine the correlation between PE severity and lipid parameters/atherogenic indices.

Main Methods:

  • A case-control study involving 96 PE patients and 84 controls.
  • Data collection included medical/sexual history, physical examination, Premature Ejaculation Diagnostic Tool (PEDT) scores, and intravaginal ejaculation latency times (IELT).
  • Calculation of atherogenic indices: Castelli Risk Index-1 (CRI-1), CRI-2, Atherogenic Index of Plasma (AIP), and Atherogenic Coefficient (AC).

Main Results:

  • Patients with PE exhibited significantly higher triglyceride and AIP values compared to controls (p=0.049 and p=0.040, respectively).
  • A fair positive correlation was observed between PEDT scores and AIP values (r=0.355, p<0.001).
  • AIP was found to be associated with both the presence of PE and its symptom severity.

Conclusions:

  • Acquired premature ejaculation is associated with adverse blood lipid parameters, specifically elevated triglycerides and AIP.
  • AIP may serve as a potential biomarker for assessing cardiovascular risk in PE patients.
  • Further research is warranted to explore the underlying mechanisms linking metabolic factors and PE.