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Incidental Asymptomatic Splenic Infarct in a COVID-19 Patient
Natasha Ghalib1, Prateek Pophali1, Natalia Chamorro-Pareja1
1Internal Medicine, Albert Einstein College of Medicine/Jacobi Medical Center, Bronx, USA.
Insights
Coronavirus disease 2019 (COVID-19) can cause arterial thrombosis, even in non-critical cases. This case highlights a silent splenic infarct in a COVID-19 patient, underscoring the need for better thromboembolism management strategies.
Area of Science:
- Cardiovascular Medicine
- Infectious Diseases
- Hematology
Background:
- Coronavirus disease 2019 (COVID-19) is associated with a high incidence of thromboembolic events and coagulation abnormalities.
- Arterial thrombosis, including in unusual locations, has been increasingly reported in patients with COVID-19.
Observation:
- A case report of a 67-year-old female with non-critical COVID-19 is presented.
- An incidental, asymptomatic splenic infarct was discovered during the patient's evaluation.
- No cardio-embolic source was identified for the splenic infarct.
Findings:
- The splenic infarct is presumed to be an arterial thromboembolic event related to COVID-19.
- This finding occurred in a patient with clinically non-critical COVID-19 disease.
- The case demonstrates a clinically silent arterial thrombotic complication of COVID-19.
Implications:
- The findings emphasize the risk of arterial thromboembolism in COVID-19 patients, even those with mild disease.
- This case highlights the ongoing debate regarding optimal anticoagulation duration and modality for COVID-19 patients.
- There is a need to refine diagnostic and management strategies for thromboembolism in COVID-19 to prevent severe complications.
Abstract:
A high incidence of thromboembolic events and coagulation parameter abnormalities are seen in cases of coronavirus disease 2019 (COVID-19). Both venous and arterial thrombosis, including arterial thrombosis in unusual sites, have been reported in COVID patients in recent literature. Herein, we report a case of a 67-year-old female patient with non-critical COVID-19 disease with an incidental finding of an asymptomatic splenic infarct. In the absence of a cardio-embolic source, we believe this was an arterial thromboembolic event in the splenic circulation. The duration and modality of anticoagulation of inpatient and ambulatory COVID patients remains a dynamic discussion. Our case adds the evidence of a clinically silent arterial thrombotic event in a non-critical COVID-19 patient which further emphasizes the need to address the strategies for diagnosis and management of thrombo-embolism to prevent potentially fatal complications.
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