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Published on: July 7, 2017
Early Interleukin-22 and Neutrophil Proteins Are Correlated to Future Lung Damage in Children With Cystic Fibrosis
Julie Renwick1, Emma Reece1, Jamie Walsh2
1Clinical Microbiology, Trinity College Dublin, Dublin, Ireland.
Insights
Early life neutrophil protein levels in Cystic Fibrosis (CF) infants may predict later lung damage. Specific proteins like azurocidin and myeloperoxidase showed positive correlations, while others like Complement C3 showed negative correlations with lung damage scores.
Area of Science:
- Pulmonary Medicine
- Biomarker Discovery
- Immunology
Background:
- Cystic Fibrosis (CF) lung damage initiates early, linked to infections and inflammation.
- Neutrophils in CF patients exhibit an altered phenotype, suggesting early dysfunction.
- Predicting disease progression is crucial for timely therapeutic interventions.
Purpose of the Study:
- To identify early life signals of dysfunctional neutrophil responses in CF.
- To determine if these early signals correlate with lung damage in later childhood.
Main Methods:
- Pulmonary protein profiles of 14 stable CF infants and pre-schoolers analyzed using SOMAscan®.
- High-resolution computed tomography (HRCT) and Brody score calculated after age 6.
- Spearman's rank correlation and Benjamini-Hochberg adjustment used for analysis.
Main Results:
- Early azurocidin and myeloperoxidase levels positively correlated with later Brody scores (p=0.0041, p=0.0182).
- Complement C3, X-ray repair CCP 6, C3a anaphylatoxin des Arginine, and cytokine receptor common subunit gamma negatively correlated with Brody scores.
- Lower early IL-22 levels were associated with more severe lung damage (p=0.0243).
Conclusions:
- Early life pulmonary protein profiles, particularly neutrophil-associated proteins, may serve as biomarkers for predicting CF lung damage progression.
- IL-22 warrants further investigation for its role in CF pathogenesis.
- Findings highlight the potential for early therapeutic interventions based on identified biomarkers.
Abstract:
Cystic Fibrosis (CF) lung damage begins early in life. Lung function decline is associated with pulmonary infections, neutrophil infiltration and inflammation. In CF, neutrophils have an altered phenotype. In this pilot study, we aimed to determine if signals of dysfunctional neutrophil responses were evident early in life and whether these signals may be associated with lung damage in later childhood. We examined the pulmonary protein profiles of 14 clinical stable infants and pre-school children with CF employing the aptamer-based affinity platform, SOMAscan®. High resolution computed tomography (HRCT) was performed on all children after age 6 years and Brody score calculated. A Spearman's rank order correlation analysis and Benjamini-Hochberg adjustment was used to correlate protein concentrations in early life to Brody scores in later childhood. Early life concentrations of azurocidin and myeloperoxidase, were positively correlated with Brody score after age 6 (p = 0.0041 and p = 0.0182, respectively). Four other neutrophil associated proteins; Complement C3 (p = 0.0026), X-ray repair CCP 6 (p = 0.0059), C3a anaphylatoxin des Arginine (p = 0.0129) and cytokine receptor common subunit gamma (p = 0.0214) were all negatively correlated with Brody scores. Interestingly, patients with more severe lung damage after age 6 had significantly lower levels of IL-22 in early years of life (p = 0.0243). IL-22 has scarcely been reported to have implications in CF. Identification of early biomarkers that may predict more severe disease progression is particularly important for the future development of early therapeutic interventions in CF disease. We recommend further corroboration of these findings in prospective validation studies.
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