[Hemodynamic changes in the blood vessels of the lesser circulation in interstitial pulmonary diseases]

Hemodynamic vascular changes of the lesser circulation in granulomatous and diffuse pulmonary diseases shown to be systemic and advanced are suggestive of the existing mechanisms underlying durable compensation of defected blood flow through alveolar septa. One of the mechanisms may be operable by means of arteriolar blood outflow into the venules along dilated major capillaries. In compliance with this conception is venous hypertrophy in tunica media and new growths of the intimal muscular layer.