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Carcinogenicity of bucetin in (C57BL/6 X C3H)F1 mice

K Togei1, N Sano, T Maeda

  • 1Second Department of Pathology, School of Medicine, University of Tokushima, Japan.

Insights

Bucetin, an antipyretic analgesic, induced renal cell tumors in male mice in a dose-dependent manner. Female mice and controls showed no kidney tumors, indicating sex-specific carcinogenicity.

Area of Science:

  • Toxicology
  • Oncology
  • Pharmacology

Background:

  • Bucetin is an antipyretic and analgesic drug.
  • Understanding the long-term effects and potential carcinogenicity of bucetin is crucial for drug safety assessment.

Purpose of the Study:

  • To evaluate the carcinogenicity of bucetin in a mouse model.
  • To determine if bucetin exposure leads to tumor development and to assess dose-response relationships.

Main Methods:

  • 300 (C57BL/6 X C3H)F1 mice (equal sexes) were fed diets containing 1.5% or 0.75% bucetin for 76 weeks, followed by 8 weeks of a basal diet.
  • Control groups received only the basal diet for 84 weeks.
  • Kidneys and other organs were examined for tumors and preneoplastic lesions.

Main Results:

  • Renal cell tumors were induced in 22% of male mice at the high dose and 13% at the low dose.
  • Dose-related dysplastic lesions of proximal tubules were frequent in male mice treated with bucetin.
  • No kidney tumors or preneoplastic lesions were observed in female mice or control animals.
  • Urinary bladder papilloma and hyperplasia were observed in some male mice at the high dose.

Conclusions:

  • Bucetin exhibits carcinogenicity in male mice, specifically inducing renal cell tumors.
  • The carcinogenic effect of bucetin on the kidney is sex-specific, with males being susceptible and females being resistant.
  • These findings highlight the importance of sex as a factor in drug-induced carcinogenicity studies.

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