[Corrigendum] Receptor for activated C kinase 1 (RACK1) promotes the progression of OSCC via the AKT/mTOR pathway

Xuefeng Zhang1, Na Liu2, Danhua Ma1

  • 1State Key Laboratory of Oral Diseases, West China Hospital of Stomatology, Sichuan University, Chengdu, Sichuan, P.R. China.

Insights

This corrigendum corrects errors in Figs. 3 and 4 regarding RACK1

Area of Science:

  • Molecular Biology
  • Oncology
  • Cancer Research

Context:

  • Original article investigated the role of RACK1 in oral squamous cell carcinoma (OSCC).
  • Figures 3 and 4 presented data on RACK1's effect on tumor markers and its correlation with p-AKT.
  • An oversight led to data presentation errors in these key figures.

Purpose:

  • To correct inadvertent data presentation errors in Figures 3 and 4 of the original publication.
  • To provide the accurate data for RACK1's role in OSCC progression and its association with p-AKT.
  • To ensure the integrity and accuracy of the scientific record.

Summary:

  • This corrigendum addresses data panel overlaps in Fig. 3 (RACK1 silencing and tumor markers) and duplication in Fig. 4 (RACK1 and p-AKT correlation).
  • Corrected versions of Figs. 3 and 4 are provided, featuring accurate data for E-cadherin experiments and RACK1/moderate dysplasia.
  • The authors confirm these corrections do not alter the original study's main conclusions.

Impact:

  • Ensures accurate representation of RACK1's function in oral squamous cell carcinoma.
  • Maintains the reliability of findings related to tumor progression markers and p-AKT signaling.
  • Upholds scientific integrity by rectifying data presentation errors for the readership.

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