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Changes in the nucleoprotein complexes of a baculovirus DNA during infection

Virology
|June 1, 1986
PubMed

Insights

Autographa californica nuclear polyhedrosis virus (AcNPV) DNA forms a chromatin-like structure in infected cells. By late infection, viral DNA adopts a unique nucleoprotein structure with viral-induced proteins.

Area of Science:

  • Molecular Virology
  • Cellular Biology
  • Epigenetics

Background:

  • Autographa californica nuclear polyhedrosis virus (AcNPV) is a baculovirus that infects insect cells.
  • Understanding viral DNA organization within host cells is crucial for comprehending viral replication and pathogenesis.

Purpose of the Study:

  • To investigate the DNA-protein complexes formed by AcNPV DNA during infection of Spodoptera frugiperda cells.
  • To characterize the nucleoprotein structure of viral DNA at different stages of infection.
  • To identify viral-induced proteins associated with viral DNA.

Main Methods:

  • Micrococcal nuclease digestion of infected nuclei.
  • Analysis of DNA fragments by gel electrophoresis.
  • Identification of viral-induced proteins using molecular weight determination.

Main Results:

  • Both parental and progeny viral DNA adopted a chromatin-like structure early in infection.
  • At late infection times (24 hr p.i.), viral DNA exhibited a unique nucleoprotein structure with mononucleosome-sized fragments (185 bp) and subnucleosomal bands (120 bp, 90 bp).
  • Two major viral-induced proteins (15K and 39K) were identified, with the 15K protein's induction correlating with the altered nucleoprotein structure and cessation of histone synthesis.

Conclusions:

  • AcNPV DNA undergoes significant structural changes during infection, forming a unique nucleoprotein complex.
  • Viral-induced proteins, particularly the 15K protein, play a role in establishing this altered viral DNA structure.
  • Host chromatin structure remains unaffected by AcNPV infection.

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