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Updated: Oct 1, 2025

Author Spotlight: Radiotherapy and Clonogenic Assays for Advancing Cancer Research and Personalized Medicine
Published on: April 5, 2024
Selective radiosensitization by nitazoxanide of quiescent clonogenic colon cancer tumour cells
Henning Karlsson1, Mårten Fryknäs1, Wojciech Senkowski1
1Department of Medical Sciences, Uppsala University, Uppsala S-751 85, Sweden.
Abstract:
Nitazoxanide is a Food and Drug Administration-approved antiprotozoal drug recently demonstrated to be selectively active against quiescent and glucose-deprived tumour cells. This drug also has several characteristics that suggest its potential as a radiosensitizer. The present study aimed to investigate the interaction between nitazoxanide and radiation on human colon cancer cells cultured as monolayers, and to mimic key features of solid tumours in patients, as spheroids, as well as in xenografts in mice. In the present study, colon cancer HCT116 green fluorescent protein (GFP) cells were exposed to nitazoxanide, radiation or their combination. Cell survival was analysed by using total cell kill and clonogenic assays. DNA double-strand breaks were evaluated in the spheroid experiments, and HCT116 GFP cell xenograft tumours in mice were used to investigate the effect of nitazoxanide and radiation in vivo. In the clonogenic assay, nitazoxanide synergistically and selectively sensitized cells grown as spheroids to radiation. However, this was not observed in cells cultured as monolayers, as demonstrated in the total cell kill assays, and much less with the clinically established sensitizer 5-fluorouracil. The sensitizing effect from nitazoxanide was confirmed via spheroid γ-H2A histone family member X staining. Nitazoxanide and radiation alone similarly inhibited the growth of HCT116 GFP cell xenograft tumours in mice with no evidence of synergistic interaction. In conclusion, nitazoxanide selectively targeted quiescent glucose-deprived tumour cells and sensitized these cells to radiation in vitro. Nitazoxanide also inhibited tumour growth in vivo. Thus, nitazoxanide is a candidate for repurposing into an anticancer drug, including its use as a radiosensitizer.
Insights
Nitazoxanide, an antiprotozoal drug, selectively sensitizes quiescent colon cancer cells to radiation in vitro. While it inhibits tumor growth in vivo, synergistic radiosensitization was not observed in animal models.
Area of Science:
- Oncology
- Pharmacology
- Cancer Research
Background:
- Nitazoxanide is an FDA-approved antiprotozoal agent with demonstrated activity against quiescent, glucose-deprived tumor cells.
- The drug exhibits characteristics suggesting potential as a radiosensitizer in cancer therapy.
Purpose of the Study:
- To investigate the interaction between nitazoxanide and radiation in human colon cancer cells.
- To evaluate this interaction in vitro using monolayers and spheroids, and in vivo using mouse xenografts.
Main Methods:
- Human colon cancer HCT116 GFP cells were treated with nitazoxanide, radiation, or combination therapy.
- Cell survival was assessed using total cell kill and clonogenic assays.
- DNA double-strand breaks and tumor growth in xenografts were evaluated.
Main Results:
- Nitazoxanide synergistically sensitized spheroid-cultured cells to radiation, an effect not seen in monolayers.
- The radiosensitizing effect was confirmed by γ-H2A histone family member X staining in spheroids.
- Nitazoxanide and radiation monotherapy inhibited xenograft tumor growth, but without synergy.
Conclusions:
- Nitazoxanide selectively targets quiescent, glucose-deprived tumor cells, enhancing their sensitivity to radiation in vitro.
- The drug demonstrates potential for repurposing as an anticancer agent and radiosensitizer.
- Further investigation into its in vivo efficacy and synergistic potential is warranted.
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