Related Experiment Video
Updated: Jun 27, 2026

09:44
Neuro-rehabilitation Approach for Sudden Sensorineural Hearing Loss
Published on: January 25, 2016
19.4K
Hearing loss after bacterial meningitis, a retrospective study
Filip Persson1, Nora Bjar2, Ann Hermansson2
1Lund University Hospital, Lund, Sweden.
Acta Oto-Laryngologica
|April 11, 2022
Summary
Bacterial meningitis can cause hearing loss, especially with concurrent acute otitis media (AOM) and pneumococcal infections. Prompt ear exams are crucial for meningitis patients to identify and manage hearing loss risks.
Area of Science:
- Otolaryngology
- Infectious Diseases
- Neurology
Background:
- Hearing loss is a frequent complication of bacterial meningitis.
- Risk factors, especially acute otitis media (AOM), are not well understood.
- This study investigates hearing loss in bacterial meningitis patients.
Purpose of the Study:
- To determine the incidence of hearing loss post-bacterial meningitis.
- To identify risk factors associated with hearing loss.
- To evaluate the role of concurrent AOM in hearing loss development.
Main Methods:
- Retrospective analysis of medical records from 2000-2017 in Skåne county, Sweden.
- Inclusion of patients treated for bacterial meningitis.
- Logistic regression used to assess risk factor associations.
Main Results:
- 187 bacterial meningitis cases identified; 71/119 patients with audiometry had hearing loss.
- Hearing loss was more prevalent in adults.
- Associations found between hearing loss and AOM, and hearing loss and pneumococcal infection.
Conclusions:
- Age, concurrent AOM, and pneumococcal infection are significant risk factors for hearing loss.
- Over a third of patients did not undergo recommended post-recovery hearing tests.
- Emphasizes the need for early ear examination and potential tympanocentesis in meningitis patients.
Related Concept Videos
Bacterial Meningitis
Bacterial meningitis is a severe infectious disease involving inflammation of the meninges, the protective membranes surrounding the brain and spinal cord. It occurs when pathogenic bacteria cross the blood–brain barrier and enter the cerebrospinal fluid. Common causative organisms include Neisseria meningitidis, Streptococcus pneumoniae, Haemophilus influenzae type b, Listeria monocytogenes, and Escherichia coli K1. The exact route of entry varies by pathogen and host condition.Routes of Entry...
Viral Meningitis
Viral meningitis is the most common form of meningitis and is often referred to as aseptic meningitis to indicate the absence of bacterial involvement. It is generally milder than bacterial meningitis, with symptoms including fever, headache, stiff neck, drowsiness, nausea, photophobia, and vomiting. Rarely, more severe manifestations or death may occur. Common causative agents include enteroviruses, particularly coxsackie A and B viruses and echoviruses, all members of the Enterovirus genus...
Bacterial Meningitis I: Introduction
Bacterial meningitis is a severe, life-threatening inflammation of the meninges, particularly the pia mater and arachnoid mater, affecting the subarachnoid space, ventricles, and cerebrospinal fluid (CSF). If untreated, it can lead to significant neurological complications or death.Causative AgentsCommon pathogens vary with age and immune status. In adults, major organisms include Streptococcus pneumoniae, Neisseria meningitidis, and Haemophilus influenzae. Streptococcus agalactiae (group B...
Bacterial Meningitis II: Pathophysiology
Bacterial meningitis typically begins when pathogens such as Neisseria meningitidis and Streptococcus pneumoniae colonize the nasopharynx and invade the bloodstream. This process is facilitated by bacterial virulence factors, such as polysaccharide capsules, which resist phagocytosis and complement-mediated killing. Less commonly, bacteria reach the central nervous system via contiguous spread from infections like otitis media or sinusitis, through congenital or acquired dural defects, or...
Brain Abscess l: Introduction
A brain abscess is a focal, intracerebral infection characterized by a localized collection of pus within the brain parenchyma, resulting from microbial invasion and the body’s inflammatory response. It progresses through stages: early and late cerebritis, followed by early and late capsule formation, reflecting tissue destruction, immune response, and eventual encapsulation.Etiology and PathogenesisCausative organisms vary with source and host factors, often involving polymicrobial infections,...

