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[Defense mechanisms of the macro-organism in bacteriuria in chronic pyelonephritis]

Zeitschrift Fur Urologie Und Nephrologie
|November 1, 1986
PubMed

Insights

This study found distinct immune responses in obstructive versus non-obstructive chronic pyelonephritis patients. Obstructive cases showed higher antibody titers and complement levels, while non-obstructive cases had altered complement and lysozyme during active disease stages.

Area of Science:

  • Nephrology
  • Immunology
  • Microbiology

Background:

  • Chronic pyelonephritis is a persistent kidney infection often caused by Escherichia coli (E. coli).
  • Understanding the host immune response is crucial for managing this condition, particularly differentiating between obstructive and non-obstructive forms.
  • Immune markers like complement, lysozyme, and antibody titers may provide insights into disease activity and patient outcomes.

Purpose of the Study:

  • To investigate and compare the immunological profiles of patients with obstructive and non-obstructive chronic pyelonephritis.
  • To assess the relationship between immune markers and disease activity in different forms of chronic pyelonephritis.
  • To evaluate the prevalence of serum bactericidal defects against homologous E. coli strains.

Main Methods:

  • Prospective study over 4 years involving 55 patients with chronic pyelonephritis.
  • Isolation and characterization of 193 E. coli strains from urine samples.
  • Measurement of serum and urine immunofluorescence titers, total complement, complement factors (C3, C4), serum and urine lysozyme, and serum bactericidal activity against homologous E. coli strains.

Main Results:

  • Patients with obstructive chronic pyelonephritis exhibited significantly higher serum immunofluorescence titers and total complement levels, and lower serum lysozyme compared to non-obstructive cases.
  • In non-obstructive pyelonephritis, active disease stages showed significantly decreased total complement and increased complement factors (C3, C4) and urine lysozyme compared to inactive stages.
  • Serum bactericidal defects against homologous E. coli were observed in 32.73% of patients, occurring equally in both obstructive and non-obstructive forms and more frequently in active disease stages.

Conclusions:

  • Distinct immunological differences exist between obstructive and non-obstructive chronic pyelonephritis, suggesting different host-pathogen interactions.
  • Immune marker fluctuations correlate with disease activity specifically in non-obstructive chronic pyelonephritis.
  • Impaired serum bactericidal activity is a notable finding in a subset of patients, irrespective of disease obstruction status.

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