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Related Concept Videos

Atherosclerosis I: Introduction01:30

Atherosclerosis I: Introduction

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Atherosclerosis is a progressive disorder characterized by the buildup of plaques on the arterial inner wall, causing them to narrow and harden over time. These plaques comprise lipids, calcium, blood components, carbohydrates, and fibrous tissue. The process primarily affects the intima of large and medium-sized arteries, reducing blood flow in any artery.Etiology and risk factorsThe cause of atherosclerosis is multifactorial, involving a complex interplay among endothelial injury, lipid...
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Peripheral Artery Disease I: Introduction01:30

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Peripheral artery disease (PAD) predominantly results from atherosclerosis, which involves the accumulation of fatty deposits, or plaques, within the walls of arteries. This causes them to narrow and harden, significantly reducing blood flow. PAD predominantly affects the legs, particularly the arteries supplying the thighs and calves. In rare cases, it may involve other arteries, including those in the arms.Etiology of PAD:The principal cause of PAD is atherosclerosis, which results from fatty...
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Atherosclerosis II: Clinical Manifestations and Diagnostic Tests01:27

Atherosclerosis II: Clinical Manifestations and Diagnostic Tests

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Atherosclerosis is a progressive disorder that leads to the thickening and narrowing of arterial walls due to plaque buildup. This condition can cause various symptoms depending on the arteries affected:Coronary Artery Disease (CAD): This condition affects the coronary arteries and may lead to chest pain (angina), shortness of breath (dyspnea), heart attacks, and other heart disease symptoms.Cerebrovascular Disease: This affects blood flow to the brain, causing transient ischemic attacks (TIAs)...
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Coronary Artery Disease II: Pathophysiology01:26

Coronary Artery Disease II: Pathophysiology

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Coronary Artery Disease (CAD) originates from a series of events that impair the function of coronary arteries, the blood vessels responsible for delivering oxygen-rich blood to the heart muscle. The pathophysiology of CAD is closely linked to atherosclerosis, a chronic inflammatory and lipid-driven condition affecting the vascular endothelium.1. Endothelial DamageThe process begins with damage to the vascular endothelium, which serves as a protective barrier between the blood and the vessel...
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Atherosclerosis IV: Nursing Management01:23

Atherosclerosis IV: Nursing Management

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Nursing management for a patient with arteriosclerosis involves a comprehensive approach focusing on lifestyle modification, disease monitoring, education, and symptomatic care. Here is an overview of effective nursing strategies:Assessment and Monitoring: Initial and ongoing assessments are crucial. Nurses must document the patient's medical history, including any hypertension, diabetes, hyperlipidemia, and other cardiovascular diseases. Assessments also cover family history and lifestyle...
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Related Experiment Video

Updated: Sep 21, 2025

A Human Ex Vivo Atherosclerotic Plaque Model to Study Lesion Biology
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[Atherosclerosis, an inflammatory disease].

A Cagnina1, O Chabot1, L Davin1

  • 1Service de Cardiologie, CHU Liège, Belgique.

Revue Medicale De Liege
|June 3, 2022
PubMed
Summary
This summary is machine-generated.

Chronic inflammation drives atherosclerosis development and complications. This article details inflammatory processes and therapeutic strategies for this cardiovascular disease.

Keywords:
Coronary diseaseInflammasomeInflammationAtherosclerosis

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Area of Science:

  • Cardiovascular Science
  • Immunology
  • Pathology

Background:

  • Chronic inflammation is a key factor in atherosclerosis, influencing its development, progression, and complications.
  • Atherosclerosis exhibits an inflammatory nature, evidenced by inflammatory cells, cytokines, and chemokines throughout disease stages.
  • Serum inflammatory markers like C-reactive protein (CRP), Interleukin-6 (IL-6), and Interleukin-1 beta (IL-1β) are linked to cardiovascular events.

Purpose of the Study:

  • To detail the involvement of inflammatory processes in atherosclerosis.
  • To review advancements in therapeutic strategies targeting inflammation in atherosclerosis.

Main Methods:

  • Review of scientific literature on inflammation and atherosclerosis.
  • Analysis of the role of specific inflammatory markers and pathways.
  • Examination of current and emerging therapeutic approaches.

Main Results:

  • Confirmation of chronic inflammation's central role in all phases of atherosclerosis.
  • Established association between cardiovascular events and elevated inflammatory markers.
  • Identification of inflammasome pathway involvement in producing key inflammatory mediators.

Conclusions:

  • Inflammation is integral to atherosclerosis pathogenesis and complications.
  • Therapeutic strategies targeting inflammatory pathways show promise for managing atherosclerosis.
  • Further research into anti-inflammatory treatments is crucial for cardiovascular health.