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COVID-19 and Aortic Thrombosis: A Case Report.
Mamtha Balla1, Hannah Staats-Ciotola2, Meera Dhavale3
1Department of Internal Medicine, University of Toledo and Promedica Toledo Hospital, Toledo, OH 43606, USA.
Summary
Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) can cause hypercoagulability, leading to blood clots like stroke and aortic thrombus in COVID-19 patients. Monitoring and anticoagulant treatment are crucial for managing this complication.
Area of Science:
- Cardiology
- Infectious Diseases
- Hematology
Background:
- Coronavirus disease 2019 (COVID-19), caused by SARS-CoV-2, primarily presents with respiratory symptoms.
- Emerging evidence suggests COVID-19 can also lead to significant hypercoagulability and thromboembolic events.
- The pandemic has highlighted the need to understand non-respiratory complications of SARS-CoV-2 infection.
Observation:
- This case report details a COVID-19 patient who experienced a stroke.
- The patient was also diagnosed with an aortic thrombus, indicating arterial thrombosis.
- The case underscores the potential for severe vascular complications in COVID-19.
Findings:
- COVID-19 patients may develop hypercoagulable states, increasing the risk of both venous and arterial thrombosis.
- Laboratory markers for hypercoagulability and symptoms of thromboembolism warrant close monitoring in infected individuals.
- Anticoagulant prophylaxis and treatment may be necessary for managing thrombotic complications.
Implications:
- Physicians should maintain a high index of suspicion for thromboembolic events in COVID-19 patients.
- Early detection and management of hypercoagulability can potentially prevent severe outcomes.
- Further research is needed to elucidate the causal relationship between SARS-CoV-2 and hypercoagulability.
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