Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Concept Videos

Esophageal Varices-I: Introduction01:24

Esophageal Varices-I: Introduction

313
Esophageal varices are dilated, tortuous veins which are found mainly in the submucosa of the lower esophagus but which may also appear higher up or extend into the stomach. They develop due to increased pressure in the portal venous system, often as a result of liver cirrhosis. This condition scars and damages the liver, impeding normal blood flow through the portal vein. To compensate, blood seeks alternative pathways, forming fragile new vessels (varices) in the esophagus and stomach. These...
313
Esophageal Varices-II: Clinical Features and Management01:28

Esophageal Varices-II: Clinical Features and Management

124
Esophageal varices often manifest as gastrointestinal bleeding episodes, presenting symptoms like hematemesis (vomiting of blood), hematochezia (passing fresh blood via the rectum), and melena (black, tarry stools). Other signs can include weight loss, anorexia, abdominal discomfort, jaundice, pruritus, altered mental status, and muscle cramps.
In the initial assessment, a thorough review of the patient's medical history is vital to identify risk factors such as liver disease, alcohol...
124
Varicose Veins I: Introduction01:26

Varicose Veins I: Introduction

26
Varicose veins, or varicosities, are abnormally dilated and twisted superficial veins caused by venous valve incompetence. This condition commonly affects the lower extremities, especially the saphenous veins, due to the higher pressure from prolonged standing and walking. However, varicosities can also occur in other areas, such as the esophagus, vulva, spermatic cords, and anorectal region.Etiology and typesPrimary varicose veins, often idiopathic, are more common in women due to inherent...
26
Varicose Veins II: Diagnostic Studies and Interprofessional Care01:26

Varicose Veins II: Diagnostic Studies and Interprofessional Care

27
Varicose veins, or varicosities, develop when the valves in the veins, which control blood flow, weaken or damage. It causes blood to pool and the veins to enlarge. Understanding the clinical manifestations, diagnostic approaches, and management options for varicose veins is crucial for effective treatment and relief.Clinical manifestationsClinical manifestations of varicose veins include a heavy, achy feeling or pain after prolonged standing or sitting. This discomfort can often be relieved by...
27
Venous Thrombosis II: Clinical Manifestations and Diagnostic Studies01:20

Venous Thrombosis II: Clinical Manifestations and Diagnostic Studies

30
The key difference between Superficial Vein Thrombosis (SVT) and Deep Vein Thrombosis (DVT) lies in their location and severity.Clinical ManifestationsSVT typically presents with localized pain, tenderness, and redness along the course of a superficial vein, often accompanied by a palpable, cord-like structure under the skin. This condition is usually less dangerous than DVT but can be uncomfortable and may lead to complications such as cellulitis or, rarely, a clot extension into the deep...
30
Veins of the Abdomen and Pelvis01:18

Veins of the Abdomen and Pelvis

913
The human body is a complex system of interconnected parts, and the circulatory system plays a crucial role in maintaining overall health. One key component of this system is the inferior vena cava, a large vein responsible for returning blood from the abdominopelvic viscera and abdominal walls to the heart.
The inferior vena cava is fed by numerous smaller veins. The lumbar veins, for instance, drain the posterior abdominal wall, emptying both directly into the inferior vena cava and into the...
913

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

Investigating the expression of CTLA-4 and CD28 genes as immune regulators in patients with colorectal polyps.

BMC cancer·2026
Same author

Early Posttransplant Lymphoproliferative Disorder and Cryptosporidiosis After Liver Transplantation.

Case reports in medicine·2026
Same author

Colon cancer versus colitis: a microRNA and gene expression analysis via system biology tools.

Gastroenterology and hepatology from bed to bench·2026
Same author

Prognostic nutritional index and systemic inflammation index as predictors of cirrhosis severity: a cross-sectional study.

Gastroenterology and hepatology from bed to bench·2026
Same author

Application of artificial intelligence in colonoscopy imaging for polyp analysis-A systematic review.

Computer methods and programs in biomedicine·2026
Same author

Integrated bioinformatics and experimental validation identify FOXQ1, GRIN2D, and SCNN1B as novel biomarkers for distinguishing high-risk sessile serrated lesions from hyperplastic polyps.

Journal of molecular histology·2026

Related Experiment Video

Updated: Sep 4, 2025

Endoscopic Injection Sclerotherapy Assisted by Cyanoacrylate and Clips for Gastroesophageal Varices
04:09

Endoscopic Injection Sclerotherapy Assisted by Cyanoacrylate and Clips for Gastroesophageal Varices

Published on: June 13, 2025

331

Transverse colon varices.

Behzad Hatami1, Naghmeh Salarieh2, Pardis Ketabi Moghadam2

  • 1Gastroenterology and Liver Diseases Research Center, Research Institute for Gastroenterology and Liver Diseases, Shahid Beheshti University of Medical Sciences, Tehran, Iran.

Gastroenterology and Hepatology From Bed to Bench
|July 18, 2022
PubMed
Summary

Ectopic colonic varices, a rare complication of liver cirrhosis, can cause severe rectal bleeding. Balloon-occluded retrograde transvenous obliteration (BRTO) effectively treated rectorrhagia in a patient with hepatitis B (HBV) cirrhosis when colonoscopic band ligation failed.

Keywords:
BRTOBand ligationColonic varices

More Related Videos

New Thrombectomy Technique for Total Portal Vein Thrombosis in Liver Transplantation
08:45

New Thrombectomy Technique for Total Portal Vein Thrombosis in Liver Transplantation

Published on: June 27, 2025

720
Underwater Endoscopic Injection Sclerotherapy for Gastroesophageal Varices
02:14

Underwater Endoscopic Injection Sclerotherapy for Gastroesophageal Varices

Published on: August 1, 2025

444

Related Experiment Videos

Last Updated: Sep 4, 2025

Endoscopic Injection Sclerotherapy Assisted by Cyanoacrylate and Clips for Gastroesophageal Varices
04:09

Endoscopic Injection Sclerotherapy Assisted by Cyanoacrylate and Clips for Gastroesophageal Varices

Published on: June 13, 2025

331
New Thrombectomy Technique for Total Portal Vein Thrombosis in Liver Transplantation
08:45

New Thrombectomy Technique for Total Portal Vein Thrombosis in Liver Transplantation

Published on: June 27, 2025

720
Underwater Endoscopic Injection Sclerotherapy for Gastroesophageal Varices
02:14

Underwater Endoscopic Injection Sclerotherapy for Gastroesophageal Varices

Published on: August 1, 2025

444

Area of Science:

  • Gastroenterology
  • Hepatology
  • Interventional Radiology

Background:

  • Liver cirrhosis commonly causes gastroesophageal varices.
  • Ectopic varices, including colonic varices, are infrequent complications of cirrhosis.
  • Rectorrhagia indicates lower gastrointestinal bleeding, potentially from colonic varices.

Observation:

  • A 75-year-old male with hepatitis B (HBV) cirrhosis presented with significant rectorrhagia.
  • Colonoscopy identified actively bleeding, tortuous colonic varices.
  • Initial endoscopic band ligation failed to achieve hemostasis.

Findings:

  • The patient underwent balloon-occluded retrograde transvenous obliteration (BRTO).
  • BRTO successfully controlled the active colonic variceal bleeding.
  • This case highlights BRTO as an effective treatment for refractory colonic variceal hemorrhage.

Implications:

  • BRTO is a viable therapeutic option for managing severe colonic variceal bleeding secondary to liver cirrhosis.
  • This approach may be particularly useful when endoscopic methods are unsuccessful.
  • Further research could explore the long-term efficacy and safety of BRTO in this patient population.