Gender disparities in multiple myeloma publications
Aala Dweik1, Hadeel Dweik1, Hira Mian2
1Department of Medicine, Faculty of Medicine University of Jordan Amman Jordan.
Ejhaem
|September 2, 2022
Summary
Gender disparities persist in multiple myeloma (MM) research publications, with females less represented, especially in senior authorship roles. Addressing these inequities is crucial for academic progress.
Area of Science:
- Hematology
- Medical Research
- Academic Publishing
Background:
- Gender disparities are a recognized issue in academic fields, often impacting female researchers disproportionately.
- Understanding these disparities within specific medical research areas is essential for promoting equity.
Purpose of the Study:
- To quantitatively analyze gender representation and authorship patterns in multiple myeloma (MM) publications.
- To identify specific areas within MM research where gender disparities are most pronounced.
Main Methods:
- A cross-sectional study design was employed.
- Analysis of 679 publications and 8898 author positions in the field of multiple myeloma.
- Comparison of authorship distribution (first, corresponding, last/senior) between male and female authors.
Main Results:
- Females constituted one-third of all authors, with a mean of 4.4 female authors per publication compared to 8.7 male authors.
- Female representation was lower in key authorship positions: 34% as first authors, 21% as corresponding authors, and 18% as last/senior authors.
- In clinical trial publications, only 17% of authors were female.
Conclusions:
- Significant gender disparities are evident in multiple myeloma publications, particularly concerning senior and corresponding authorship.
- These findings highlight the need for targeted interventions to address and mitigate gender-based inequities in scientific publishing.
- Further investigation into the underlying factors contributing to these disparities is recommended to foster a more inclusive research environment.
Related Concept Videos
Cancer
49.4K
Cancers arise due to mutations in genes involved in the regulation of cell division, which leads to unrestricted cell proliferation. Modern science and medicine have made great strides in the understanding and treatment of cancer, including eradicating cancer in some patients. However, there is still no cure for cancer. This is largely due to the fact that cancer is a large group of many diseases.
49.4K
Sex-linked Disorders
102.7K
Like autosomes, sex chromosomes contain a variety of genes necessary for normal body function. When a mutation in one of these genes results in biological deficits, the disorder is considered sex-linked.
102.7K
Multiple Allele Traits
34.6K
The Concept of Multiple Allelism
34.6K
Nondisjunction
76.1K
During meiosis, chromosomes occasionally separate improperly. This occurs due to failure of homologous chromosome separation during meiosis I or failed sister chromatid separation during meiosis II. In some species, notably plants, nondisjunction can result in an organism with an entire additional set of chromosomes, which is called polyploidy. In humans, nondisjunction can occur during male or female gametogenesis and the resulting gametes possess one too many or one too few chromosomes.
76.1K
Bias in Epidemiological Studies
543
Biases can arise at various stages of research, from study design and data collection to analysis and interpretation. Recognizing and addressing these biases is essential to ensure the validity and reliability of epidemiological findings.Broadly speaking, biases in epidemiology fall into three main categories: selection bias, information bias, and confounding. A more detailed description of possible biases is:
543
X-linked Traits
55.2K
In most mammalian species, females have two X sex chromosomes and males have an X and Y. As a result, mutations on the X chromosome in females may be masked by the presence of a normal allele on the second X. In contrast, a mutation on the X chromosome in males more often causes observable biological defects, as there is no normal X to compensate. Trait variations arising from mutations on the X chromosome are called “X-linked”.
55.2K


