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Optic pathway conduction in insulin-dependent diabetics.
Documenta Ophthalmologica. Advances in Ophthalmology
|November 15, 1986
Summary
Diabetic patients without retinopathy show subclinical damage via abnormal visual evoked cortical potentials (VECPs) latency. On-off VECP latency alterations in diabetes, similar to pattern-reversal VECPs, can identify at-risk individuals.
Area of Science:
- Neuroscience
- Ophthalmology
- Endocrinology
Background:
- Subclinical visual pathway damage in diabetes is indicated by abnormal pattern-reversal visual evoked cortical potentials (VECPs) latency.
- Pattern-reversal VECPs and onset-offset (on-off) VECPs may originate from different cortical levels.
Purpose of the Study:
- To investigate on-off VECP latencies in insulin-dependent diabetics without retinopathy or neuropathy.
- To correlate on-off VECP latencies with clinical and metabolic parameters in diabetic patients.
Main Methods:
- Studied 50 insulin-dependent diabetic patients without retinopathy, neuropathy, or abnormal oscillatory potentials.
- Measured and analyzed on-off VECP latencies.
- Correlated VECP findings with clinical and metabolic data.
Main Results:
- Diabetic patients exhibited on-off VECP latency alterations, mirroring pattern-reversal VECP findings but to a lesser degree.
- These latency changes suggest subclinical damage in the visual pathways of diabetics.
Conclusions:
- On-off VECP latency alterations are present in diabetes, similar to pattern-reversal VECPs.
- On-off VECP latency can serve as a valuable index for assessing patients at risk of diabetic complications.