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Updated: Jul 27, 2026

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Normothermic Cardiac Arrest and Cardiopulmonary Resuscitation: A Mouse Model of Ischemia-Reperfusion Injury
Published on: August 30, 2011
Polyuria and refractory hypernatremia after cardiopulmonary arrest
The American Journal of Medicine
|February 1, 1987
Summary
A patient developed severe hypernatremia after cardiac arrest, unresponsive to vasopressin. The condition resolved with furosemide and free water, indicating complex osmotic dysregulation.
Area of Science:
- Nephrology
- Endocrinology
- Cardiology
Background:
- Myocardial infarction and cardiopulmonary arrest can precipitate complex electrolyte disturbances.
- Refractory hypernatremia suggests significant osmotic homeostasis impairment.
- Vasopressin deficiency or resistance is a key consideration in hypernatremia management.
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