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Published on: May 24, 2021
Two Strokes in One Patient: An Interesting Case Report
Abhay Lune1, Apurva Prabhudesai1
1Ophthalmology, Dr. D. Y. Patil Medical College, Hospital and Research Centre, Dr. D. Y. Patil Vidyapeeth (Deemed to be University), Pune, IND.
Insights
Transient visual blurring can precede central retinal artery occlusion (CRAO), a serious condition causing sudden vision loss. Recognizing these warning signs and managing risk factors are crucial for preventing CRAO.
Area of Science:
- Ophthalmology
- Neurology
- Vascular Medicine
Background:
- Central retinal artery occlusion (CRAO) is a critical ophthalmic emergency leading to profound vision loss.
- Transient visual disturbances preceding CRAO are often overlooked but can signify impending ischemia.
Observation:
- A 50-year-old male presented with CRAO in his left eye.
- He reported a history of transient visual blurring 15 days prior, a past stroke, discontinued anticoagulation, chronic smoking, and poorly controlled diabetes mellitus.
Findings:
- Funduscopic examination revealed macular edema with a cherry-red spot and blurred optic disc margins.
- Prompt treatment including ocular massage and paracentesis improved vision to finger counting at 2 meters.
Implications:
- This case underscores the significance of transient ischemic symptoms as precursors to CRAO.
- Maintaining long-term anticoagulant therapy and strict control of risk factors like diabetes and smoking are vital for preventing CRAO.
Abstract:
Central retinal arterial occlusion (CRAO) causes a sudden and devastating visual loss. Transient blurring of vision may precede CRAO and is often ignored by the patient, as it may resolve spontaneously without permanent visual loss. However, this can be a warning sign of ischaemia, particularly in individuals with multiple risk factors. We report a case of a 50-year-old man with CRAO in his left eye. The patient had a history of transient blurring of vision in the same eye 15 days prior, which resolved without intervention. He also had a history of stroke 12 years ago and had been on anticoagulants but discontinued them three years before this episode. This, combined with chronic smoking and poorly controlled diabetes mellitus, likely contributed to a hypercoagulable state, leading to thrombus formation responsible for CRAO. Fundus examination revealed an opaque and oedematous macula with a cherry red spot and blurred optic disc margins. Treatment with ocular massage and paracentesis improved the patient's vision to finger counting at 2 m. The patient was started on aspirin and advised of strict blood sugar control. This case highlights the importance of recognising transient ischaemic symptoms and maintaining long-term anticoagulant therapy to prevent severe complications like CRAO.
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