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Related Concept Videos

Glaucoma: Overview01:25

Glaucoma: Overview

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Glaucoma is an eye condition characterized by increased intraocular pressure that damages the retina and optic nerve, leading to irreversible blindness if left untreated. The human eye has various components, including the cornea, iris, pupil, lens, and optic nerve. Aqueous humor is secreted by the epithelium of the ciliary body in the posterior chamber and flows through the trabecular meshwork and canal of Schlemm, maintaining normal intraocular pressure. The trabecular meshwork and the canal...
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Open Angle Glaucoma: Treatment01:27

Open Angle Glaucoma: Treatment

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In open-angle glaucoma, the iridocorneal angle remains open, but the trabecular meshwork becomes stiff, slowing down the outflow of aqueous humor. This causes a buildup of aqueous humor in the anterior chamber, leading to a sudden increase in intraocular pressure. The treatment for open-angle glaucoma focuses on reducing the elevated intraocular pressure by either decreasing the secretion of aqueous humor or increasing its outflow.
Drugs such as carbonic anhydrase inhibitors, α2- and...
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Angle Closure Glaucoma: Treatment01:28

Angle Closure Glaucoma: Treatment

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Angle-closure glaucoma, or closed-angle glaucoma, is an eye condition where the iris bulges out and blocks the iridocorneal angle, resulting in a buildup of aqueous humor and increased intraocular pressure. Immediate medical attention is necessary due to the sudden onset of symptoms. The treatment for angle-closure glaucoma includes short-term and long-term approaches. Short-term treatment involves using eye drops like pilocarpine to lower intraocular pressure by increasing aqueous humor...
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Related Experiment Video

Updated: Jun 10, 2025

Assessing Early Stage Open-Angle Glaucoma in Patients by Isolated-Check Visual Evoked Potential
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Central visual field in glaucoma: An updated review.

Kelvin H Du1, Alireza Kamalipour1, Sasan Moghimi1

  • 1Viterbi Family Department of Ophthalmology, Hamilton Glaucoma Center, Shiley Eye Institute, University of California, San Diego, CA, USA.

Taiwan Journal of Ophthalmology
|October 21, 2024
PubMed
Summary

The 10-2 visual field (VF) test offers valuable insights into central vision for glaucoma patients. This study identifies patient characteristics that increase the likelihood of central VF damage, aiding in earlier detection and management.

Keywords:
Artificial intelligencecentral visual fieldglaucomaquality of life

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Area of Science:

  • Ophthalmology
  • Neuroscience

Background:

  • Central vision is crucial for daily activities, and glaucoma significantly impacts it.
  • The 10-2 visual field (VF) test is vital for assessing central vision in glaucoma but may be underutilized.
  • Current standard 24-2 VF testing might miss subtle central vision deficits.

Purpose of the Study:

  • To identify patient characteristics associated with a higher likelihood of central visual field damage.
  • To evaluate the clinical utility of the 10-2 VF test in specific glaucoma patient subgroups.
  • To assess the benefits of integrating the 10-2 VF test into routine clinical practice.

Main Methods:

  • Retrospective analysis of patient data including visual field test results.
  • Comparison of 10-2 VF and 24-2 VF test outcomes.
  • Statistical analysis to identify predictors of central VF damage.

Main Results:

  • Specific patient factors were identified that correlate with increased risk of central VF damage.
  • The 10-2 VF test provided complementary information in various clinical scenarios, including advanced glaucoma and specific ocular conditions.
  • The study highlights the potential for earlier detection of central vision loss using the 10-2 VF pattern.

Conclusions:

  • The 10-2 VF assessment is beneficial for comprehensive central vision evaluation in glaucoma.
  • Incorporating the 10-2 VF test can enhance glaucoma diagnosis, progression monitoring, and management, especially in at-risk populations.
  • Further research, potentially involving AI, could optimize the use of VF testing for resource allocation.