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Lack of experimental vesicant activity for the anticancer agents cisplatin, melphalan, and mitoxantrone

Insights

Cisplatin, L-PAM, and mitoxantrone chemotherapy drugs were evaluated for skin damage potential. Studies found these anticancer agents are not vesicants at clinical doses.

Area of Science:

  • Oncology
  • Dermatology
  • Pharmacology

Background:

  • Cisplatin and L-PAM are DNA-crosslinking anticancer agents.
  • Mitoxantrone is an anthracene-based DNA intercalator used in cancer treatment.
  • Systematic study of vesicant potential for these agents was lacking.

Purpose of the Study:

  • To evaluate the vesicant potential of cisplatin, L-PAM, and mitoxantrone.
  • To determine if these anticancer drugs cause skin necrosis at clinical doses.

Main Methods:

  • Dehaired BALB/c mice received intradermal injections of cisplatin, L-PAM, or mitoxantrone.
  • Doses administered were equivalent to human clinical doses.
  • Skin sites were monitored for necrosis and ulceration.

Main Results:

  • Cisplatin (up to 150 mg/m2) and L-PAM (up to 71 mg/m2) did not cause skin necrosis.
  • Mitoxantrone (up to 14 mg/m2) was not ulcerogenic, but caused temporary blue skin discoloration.
  • The L-PAM solvent alone was ulcerogenic when undiluted.

Conclusions:

  • Cisplatin, L-PAM, and mitoxantrone are not vesicants at clinically relevant doses.
  • The solvent for L-PAM may possess ulcerogenic properties.
  • Further investigation into drug-specific tissue reactions is warranted.

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