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Related Concept Videos

Inflammatory Bowel Disease III: Diagnostic Studies and Management I-Nutritional Therapy01:30

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Various diagnostic tests are employed in the diagnostic process for Inflammatory Bowel Disease (IBD), particularly to differentiate between Crohn's disease and ulcerative colitis.
Diagnostic studies
A colonoscopy is the definitive screening test, distinguishing ulcerative colitis from other colon diseases with similar symptoms. During a colonoscopy test, inflamed mucosa with exudate ulcerations can be observed, and biopsies are taken to determine the histologic characteristics of the...
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Gastritis III: Clinical Manifestations and Management01:23

Gastritis III: Clinical Manifestations and Management

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The clinical manifestations of gastritis can vary depending on the cause and type of gastritis, but some common symptoms may include the following.
Clinical manifestations of acute gastritis
The patient with acute gastritis may have a rapid onset of symptoms, such as epigastric pain or discomfort, dyspepsia, anorexia, hiccups, or nausea and vomiting, which can last from a few hours to a few days. Erosive or hemorrhagic gastritis may cause bleeding, which may manifest as blood in vomit or as...
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Overview of Protein Metabolism01:21

Overview of Protein Metabolism

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Proteins are broken down into amino acids during digestion. Unlike fats and carbohydrates, which are stored for later use, proteins are not. Instead, amino acids are either used to produce ATP through oxidation or contribute to the creation of new proteins for the growth and repair of the body. Any surplus amino acids from the diet are converted into glucose or triglycerides rather than excreted.
Amino acids play various roles in the body once they are absorbed into cells. They are restructured...
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Gastritis-II: Pathophysiology01:17

Gastritis-II: Pathophysiology

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Gastritis is marked by disruption of the mucosal barrier that usually protects the stomach tissue from digestive juices and manifests in acute and chronic forms.
In acute gastritis, the gastric mucosa becomes swollen and red and undergoes superficial erosion. Superficial ulceration may lead to bleeding.
In chronic gastritis, persistent or repeated insults lead to chronic inflammatory changes and, eventually, thinning or atrophy of the gastric tissue.
Gastritis can stem from various causes, each...
259
Gastritis-I: Introduction and Types01:27

Gastritis-I: Introduction and Types

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Gastritis, defined by the inflammation or irritation of the stomach lining or gastric mucosa, manifests in several distinct forms: acute, chronic, reactive, and a specific subtype known as autoimmune metaplastic atrophic gastritis.
Acute gastritis presents as a sudden inflammation triggered by various stressors to the stomach lining, such as exposure to corrosive agents, local irritants like aspirin and other NSAIDs, alcohol consumption, radiation therapy, physical trauma, severe burns, sepsis,...
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Peptic Ulcer Disease IV: Management01:26

Peptic Ulcer Disease IV: Management

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Medical treatment strategies for peptic ulcers encompass various methods. The primary goal of treatment is to diminish gastric acidity and strengthen mucosal defense mechanisms.
The therapeutic approach involves ensuring adequate rest, implementing drug therapy, promoting smoking cessation, making dietary modifications, and emphasizing long-term follow-up care.
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Association of Chronic Pain with Biomarkers of Neurodegeneration, Microglial Activation, and Inflammation in Cerebrospinal Fluid and Impaired Cognitive Function.

Annals of neurology·2023
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[Update Gout].

Therapeutische Umschau. Revue therapeutique·2023
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The target uric acid level in multimorbid patients with gout is difficult to achieve: data from a longitudinal Swiss single-centre cohort.

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Related Experiment Video

Updated: Jun 8, 2025

Glycemic Impact on Knee Osteoarthritis Symptoms on Physical, Radiographic, and Inflammatory Markers among Individuals Aged 50 and Over with Diabetes
07:22

Glycemic Impact on Knee Osteoarthritis Symptoms on Physical, Radiographic, and Inflammatory Markers among Individuals Aged 50 and Over with Diabetes

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[Gout and Nutrition].

Barbara Ankli1,2,3

  • 1Rheumazentrum Basel Dr. med. B. Ankli, Basel.

Therapeutische Umschau. Revue Therapeutique
|November 7, 2024
PubMed
Summary

Dietary changes, particularly a plant-based diet, can help manage hyperuricemia and reduce gout flares. This approach complements drug therapy for achieving target serum uric acid (SUA) levels and improving cardiovascular health.

Area of Science:

  • Rheumatology
  • Nutrition Science
  • Metabolic Health

Background:

  • Gout is a prevalent arthritis strongly associated with cardiovascular disease and metabolic syndrome.
  • Hyperuricemia, elevated serum uric acid levels, is the primary risk factor for gout.
  • Dietary factors, including purine metabolism and gut microbiome, influence serum uric acid levels.

Purpose of the Study:

  • To evaluate the role of dietary adjustments in managing hyperuricemia and gout.
  • To explore the impact of a plant-based diet on serum uric acid levels and gout flares.
  • To assess the contribution of dietary changes alongside drug therapy for gout management.

Main Methods:

  • Review of current literature on gout, hyperuricemia, and dietary interventions.

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  • Analysis of the relationship between purine metabolism, gut microbiome, and serum uric acid.
  • Evaluation of the effectiveness of plant-based diets versus traditional purine restriction.

    • Main Results:

    • A shift towards plant-based diets is more effective than simple purine restriction for managing gout.
    • Dietary changes can positively influence serum uric acid levels and decrease gout flare frequency.
    • While alcohol reduction is beneficial, it has a limited impact on serum uric acid levels.

    Conclusions:

    • Dietary modification, especially a plant-based approach, is a crucial component in managing hyperuricemia and gout.
    • Integrating dietary changes with pharmacotherapy can accelerate achieving target serum uric acid levels.
    • Patient education on diet empowers individuals to actively manage their condition and reduce gout flares.