Acute lower limb ischemia revealing hypo granular acute promyelocytic leukemia

Sabrina Belmahi1,2, Zainab Kajeiou1,2, Loubna Yacoubi1,2

  • 1Hematology Laboratory, Central laboratory, Mohammed VI University Hospital, Oujda, Morocco.

Leukemia Research Reports
|December 18, 2024
PubMed
Abstract

Insights

Lower limb ischemia revealed a rare case of hypogranular acute promyelocytic leukemia (AML-M3) in a child. This presentation highlights a unique diagnostic pathway for this aggressive hematologic malignancy.

Area of Science:

  • Hematology
  • Pediatric Oncology
  • Vascular Medicine

Background:

  • Acute promyelocytic leukemia (AML-M3), now classified as acute myeloid leukemia with PML-RARA, is characterized by abnormal promyelocyte proliferation.
  • It is associated with significant risks of bleeding and thromboembolic complications, often presenting in adults.
  • The hypogranular variant (AML3v) is particularly rare.

Observation:

  • An 11-year-old child presented with severe lower limb ischemia following minor ankle trauma.
  • Diagnostic workup revealed subtotal femoral artery thrombosis, hyperleukocytosis, thrombocytopenia, anemia, and 88% blasts.
  • Karyotyping confirmed the characteristic t(15;17) translocation, diagnosing acute myeloid leukemia (AML-M3).

Findings:

  • This case represents a rare instance of hypogranular acute promyelocytic leukemia being diagnosed through an ischemic event in a pediatric patient.
  • The underlying mechanism of ischemia in APL can involve blast cell obstruction or a hypercoagulable state.
  • Diagnosis requires a combination of clinical, morphological, phenotypic, and cytogenetic evaluations.

Implications:

  • This case underscores the importance of considering hematologic malignancies in pediatric patients presenting with unusual vascular events like lower limb ischemia.
  • Early diagnosis and prompt initiation of chemotherapy, potentially including all-trans retinoic acid (ATRA) and arsenic trioxide (ATO), are crucial for favorable outcomes.
  • Further research into the pathogenesis of thromboembolic events in APL may improve management strategies.