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Intergluteal fold depth has no influence on pilonidal sinus disease development.

Matthias Maak1,2, Philipp Mörsdorf3, Layla Bari4

  • 1Department of Surgery, University Hospital Erlangen of the Friedrich-Alexander University Erlangen-Nuremberg, Erlangen, Germany.

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|March 28, 2025
PubMed
Summary

Intergluteal fold depth is not a risk factor for pilonidal sinus disease (PSD). This study found no significant difference in fold depth between PSD patients and controls, challenging previous theories. Further research is needed to understand PSD etiology.

Keywords:
Intergluteal fold depthPSDmechanism of diseasenatal cleftpilonidal sinus

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Area of Science:

  • Medical Research
  • Surgical Pathology
  • Anatomy

Background:

  • The exact cause of primary pilonidal sinus disease (PSD) is unknown.
  • Previous studies suggested a link between deeper natal clefts and PSD.
  • The role of intergluteal fold (IGF) depth in PSD development requires clarification.

Purpose of the Study:

  • To investigate the association between intergluteal fold (IGF) depth and the risk of developing pilonidal sinus disease (PSD).
  • To determine if IGF depth is a contributing factor to PSD etiology.

Main Methods:

  • A prospective clinical study involving 95 patients with PSD and 105 controls.
  • Standardized five-step protocol used to measure IGF depth from glabella sacralis to the anus.
  • Measurements were conducted on individuals from a northern German population.

Main Results:

  • Mean IGF depth increased from the sacral glabella towards the anus, with the deepest point at the anus.
  • No statistically significant difference in IGF depth was observed between PSD patients and non-PSD individuals.
  • PSD predominantly occurred in the proximal (cranial) third of the IGF, irrespective of maximum depth.

Conclusions:

  • Intergluteal fold (IGF) depth does not appear to be a significant risk factor for primary pilonidal sinus disease (PSD).
  • The findings suggest that other factors are more likely responsible for the development of PSD.
  • Further investigation into the multifactorial etiology of PSD is warranted.