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Infection and transformation of mouse cells by human adenovirus type 2

Virology
|November 1, 1985
PubMed

Insights

Mouse cells are less efficient at replicating adenovirus type 2 than rat cells, suggesting a late-stage defect in virus production. Establishing adenovirus-transformed mouse cell lines proved exceptionally difficult.

Area of Science:

  • Virology
  • Cell Biology
  • Molecular Biology

Background:

  • Adenovirus type 2 infects various cell types, including rodent fibroblasts.
  • Susceptibility to viral infection and transformation can vary significantly between cell lines and species.

Purpose of the Study:

  • To compare the permissiveness of mouse embryo fibroblasts (MEF) and baby mouse kidney (BMK) cells to adenovirus type 2 infection and transformation versus rat embryo fibroblasts (REF).
  • To investigate potential differences in adenovirus replication, antigen production, and transformation efficiency between mouse and rat cells.

Main Methods:

  • Infection of MEF, BMK, and REF cells with adenovirus type 2.
  • Assay of viral replication and production of viral tumor and structural antigens.
  • Transfection of adenovirus type 2 or 5 DNA into cells to assess transformation frequencies.
  • Attempting to establish permanent adenovirus-transformed cell lines.

Main Results:

  • MEF and BMK cells were approximately 10-fold less permissive to adenovirus type 2 replication than REF cells.
  • Similar percentages of all cell types produced viral antigens, indicating a late-stage defect in mouse cells.
  • Transformation frequencies were comparable across cell types, but establishing permanent transformed mouse cell lines was highly challenging (2/100+ attempts).
  • Established transformed MEF lines retained and expressed viral genetic information.

Conclusions:

  • Mouse cells exhibit a defect in a late stage of adenovirus type 2 replication, likely assembly or maturation.
  • While adenovirus can transform mouse cells, establishing permanent transformed lines is inefficient compared to rat cells.
  • Further research is needed to elucidate the specific molecular mechanisms underlying these species-specific differences in adenovirus replication and transformation.

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